Nursing Care and Pathophysiology for Heart Failure (CHF)

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Nichole Weaver
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Included In This Lesson

Study Tools For Nursing Care and Pathophysiology for Heart Failure (CHF)

Heart Failure-Origin (Mnemonic)
Heart Failure – Right Sided (Mnemonic)
Heart Failure-Left-Sided (Mnemonic)
Heart Failure Pathochart (Cheatsheet)
R v L Heart Failure (Cheatsheet)
Pitting Edema (Image)
Jugular Venous Distension (Image)
Heart Failure CHF Cardiac (Image)
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Outline

 

Pathophysiology: In heart failure, the heart does not pump effectively. This can occur because of many reasons but usually, because there has been damage to the heart tissue. The heart is not able to pump enough fluid forward so fluid then backs up. This fluid backup increases work on the heart as it tries to keep up and cannot.


Overview

The heart is a pump, circulates blood throughout the body. Heart failure = pump failure. Heart failure occurs when the heart cannot pump enough blood to supply the body’s needs.


Nursing Points

General

  1. Pump Failure
    1. Decreased perfusion forwards
    2. Increased congestion backwards
  2. Causes
    1. Myocardial Infarction
      1. Dead muscle can’t pump
    2. Hypertension
      1. ↑ afterload = ↑ stress on heart muscle
    3. Valve Disorders
      1. Blood not moving in right direction
      2. Inefficient pump
  3. Diagnostics
    1. BNP (Brain Natriuretic Peptide) – stretch of LV
    2. Echocardiogram
      1. Ejection Fraction
      2. Can diagnose valve disorder
    3. Chest X-Ray (CXR
      1. Cardiomegaly
      2. Pulmonary Edema
  4. Complications
    1. Volume Overload
      1. Pulmonary Edema
      2. Exacerbations
    2. Decreased Perfusion
      1. Heart
        1. Angina, MI
        2. Arrhythmias
      2. Organs
        1. Impaired Kidney Function

Assessment

  1. Right-Sided Heart Failure
    1. Decreased Pulmonary Perfusion
      1. ↓ oxygenation
      2. ↓ activity tolerance
    2. Increased Systematic Congestion
      1. Peripheral Edema
      2. ↑ JVD
      3. ↑ Preload
      4. Weight Gain
      5. Fatigue
      6. Liver / GI Congestion
  2. Left Sided Heart Failure
    1. Decreased Systemic Perfusion
      1. Skin pale or dusky
      2. ↓ Peripheral pulses
      3. Slow  capillary refill
      4. ↓ renal perfusion
        1. ↓ urine output
        2. Kidney Injury / Failure
    2. Increased Pulmonary Congestion
      1. Pulmonary edema
        1. Cough
        2. Pink/frothy sputum
        3. Crackles
        4. Wheezes
        5. Tachypnea
        6. SOB on Exertion
      2. Anxiety/restlessness

Therapeutic Management

Goal is to decrease workload on heart while still increasing cardiac output. Discussed in more detail in Therapeutic Management Lesson

  1. Decrease Preload
  2. Decrease Afterload
  3. Increase Contractility

Patient Education

Discussed in more detail in Therapeutic Management Lesson

  1. Diet & Lifestyle Changes
  2. Medication Instructions
  3. Activity Restrictions
  4. Frequent Follow-Ups

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ADPIE Related Lessons

Related Nursing Process (ADPIE) Lessons for Nursing Care and Pathophysiology for Heart Failure (CHF)

Transcript

This lesson is going to cover the basics of heart failure.

Heart disease is one of the leading causes of death in the US so you will see Heart Failure quite frequently in clinical practice. We want you to get an Ah-hah moment here and really understand this stuff. We’re gonna give you the most important things you need to know.

So, what exactly causes heart failure? Well remember it’s just pump failure – so anything that impacts the heart’s ability to pump efficiently can cause heart failure.

The top two causes are MI’s and hypertension, but valve disorders can cause a lot of problems, too. So what happens here? Well in an MI, if you remember from those lessons, blood supply is cut off to the heart muscle and the muscle itself dies. Muscle that is dead can’t contract, so the pumping ability or contractility of the heart is affected. Hypertension causes an increased afterload – remember from the hemodynamics lesson.

The heart has to pump harder against a higher pressure in the vessels. After a while, it can get worn out. Ultimately the ventricles can’t efficiently pump against that pressure any more. Someone with valve disorders, like stenosis or regurgitation, will have issues getting the blood where it’s supposed to be. If it’s not going where it’s supposed to, then it’s backing up – causing increased congestion or stretch or preload in the heart. This stress on the heart can lead to heart failure. So again, anything that impacts the heart’s ability to pump efficiently can cause heart failure – this means anything that affect the heart muscle.

BNP Heart Failure Guidelines

So there are multiple ways that we can diagnose and classify heart failure, but we just want you to know these three. The lab value we use is called Brain Natriuretic Peptide or BNP – this is released whenever the ventricles are stretched. So in Congestive Heart Failure patients when they’re severely volume overloaded, we can see this number jump into the thousands. In the labs course we will talk in much more detail about this value, so be sure to check that out.

Heart Failure Chest X-Ray

We’ll also do a chest x-ray, which could show a couple things. You may see the heart is actually enlarged because of the overload and stretch in the ventricles – and you may also see fluid in the lungs – that’s the pulmonary edema. We’ll also get an echocardiogram which can tell us things like end-diastolic volumes (remember that measures preload), ejection fraction, which is usually less than 50% in heart failure, and whether or not there are any valve issues.

Clinical Signs of Heart Failure

So let’s look at what this patient looks like when you actually see them in practice…

In the module intro we asked y’all to brainstorm what you thought this patient looked like. Guys if you get nothing else about Heart Failure, THIS is the part you’ve got to get.

Remember we said that there’s decreased perfusion forward and increased congestion backwards, right? So in right-sided heart failure we’re going to see some oxygenation problems because of decreased perfusion to the lungs. They may struggle with activity because they just don’t have enough blood flow for their lungs!

Then backwards we see that congestion into the system – they are way overloaded in the systemic circulation – so what does that mean? That means peripheral edema – okay how would you feel if you swelled up like crazy? You’d be tired, you’d probably gain some weight? Some of that fluid may even collect in the gut and make you nauseous.

Then because the blood can’t get past the heart from the body, it even backs up into the neck and you’ll see this crazy jugular venous distention – it looks like a rope on their neck!! So if you’re seeing these signs of excessive volume out in the body – you gotta think right sided heart failure!

Okay, so what about the left? Well they actually have decreased perfusion to the body. Remember your signs of decreased perfusion? They’re gonna be pale, decreased pulses, maybe slow cap refill, and their skin might even be cold, right? Signs of decreased perfusion. Then, we see the congestion happening in their lungs – y’all their lungs are full of fluid! How would you feel? You’d be struggling to breathe, right? They’ll have a cough, and their sputum will be pink and frothy because there’s just so much blood pumping through the lungs! Imagine if this was you and had all this fluid in your lungs – what position would you want to be in? You lay down and you’ll feel like you’re drowning – a lot of these patients sleep with lots of pillows or even in a recliner. Some of them even lose weight because it becomes a choice to either eat or breathe! So if you see these severe respiratory issues and fluid on the lungs, you’ve got to think Left-sided heart failure!

So right sided the classic sign is the systemic overload and left-sided classic sign is pulmonary edema. If you get this you can pick out any issues they’ll have!

Complications of Heart Failure

Now, there are quite a few other complications of heart failure, but the one I really want y’all to understand is what happens when the kidneys don’t get perfused. So here’s our heart failure patient who has decreased perfusion forward and increased congestion backward. They’re volume overloaded, probably struggling to breathe, and now, they aren’t perfusing their kidneys.

When the kidneys lose blood flow, it stimulates the Renin-Angiotensin-Aldosterone system (RAAS). You will learn more about this in pharmacology, but what you need to know is that it causes three main things to happen in the body.

  1. The first is water retention because of aldosterone and ADH. The kidneys see a lack of flow and think they need to hold onto water! So it is increasing the preload (or stretch) on ventricles whose preload is already sky-high!
  2. The second is vasoconstriction. This is the body trying to pull blood towards the heart to increase the blood pressure – this increases afterload (the force the heart has to pump against) – in a patient whose heart is already struggling as it is!
  3. And the third is the RAAS activates the Sympathetic Nervous System – it’s basically telling the heart to work harder and faster – which it cannot do! The end result is MORE volume overload, MORE stress on the heart muscle, and a perpetuated cycle that never ends.

That’s why I call it the cycle of death. What you’ll see when we look at therapeutic management in the next lesson is that the majority of therapy is aimed at breaking this cycle.

So let’s recap – anything that can affect the heart’s ability to pump effectively can cause heart failure, including an MI, hypertension, and valve disorders. Right-sided heart failure presents with symptoms of decreased pulmonary perfusion and increased systemic congestion – so they’re swollen, gain weight, lots of edema. Left-sided is the opposite – decreased systemic perfusion and increased pulmonary congestion, so these patients are really struggling to breathe. We diagnose with a BNP, Chest X-ray, and Echocardiogram. And don’t forget that the impact this has on the kidneys can make the problem worse – we’ll talk about how we work to break that cycle of death for these patients.

I hope we’ve been able to clear this up for you a bit, like we said sometimes heart failure is overcomplicated! Our goal is to make it easier to understand to give you that peace of mind and confidence! So go out and be your absolute best self today and, as always, happy nursing!

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Final Exam

Concepts Covered:

  • Terminology
  • Urinary System
  • Respiratory Disorders
  • Acute & Chronic Renal Disorders
  • Disorders of the Adrenal Gland
  • Oncology Disorders
  • Integumentary Disorders
  • Preoperative Nursing
  • Musculoskeletal Trauma
  • Integumentary Disorders
  • Respiratory Emergencies
  • Disorders of the Posterior Pituitary Gland
  • Hematologic Disorders
  • Renal Disorders
  • Labor Complications
  • Immunological Disorders
  • Upper GI Disorders
  • Neurological Emergencies
  • Disorders of Pancreas
  • Musculoskeletal Disorders
  • Cardiac Disorders
  • Disorders of the Thyroid & Parathyroid Glands
  • Integumentary Important Points
  • Pregnancy Risks
  • Urinary Disorders
  • Vascular Disorders
  • Central Nervous System Disorders – Brain
  • Nervous System
  • Lower GI Disorders
  • Intraoperative Nursing
  • Eating Disorders
  • Circulatory System
  • Postoperative Nursing
  • Liver & Gallbladder Disorders
  • Emergency Care of the Cardiac Patient
  • Female Reproductive Disorders
  • Shock
  • Respiratory System
  • Substance Abuse Disorders
  • Fetal Development
  • Proteins
  • Noninfectious Respiratory Disorder
  • Newborn Care
  • Statistics
  • Emergency Care of the Neurological Patient
  • Basics of Sociology
  • Bipolar Disorders
  • Infectious Respiratory Disorder

Study Plan Lessons

Diagnostic Testing Course Introduction
Fluid & Electrolytes Course Introduction
X-Ray (Xray)
X-Ray (Xray)
X-Ray (Xray)
Nursing Care and Pathophysiology of Acute Kidney (Renal) Injury (AKI)
Addisons Disease
Computed Tomography (CT)
Computed Tomography (CT)
Computed Tomography (CT)
Fluid Pressures
Informed Consent
Nursing Care and Pathophysiology for Cushings Syndrome
Fluid Shifts (Ascites) (Pleural Effusion)
Magnetic Resonance Imaging (MRI)
Magnetic Resonance Imaging (MRI)
Magnetic Resonance Imaging (MRI)
Preoperative (Preop)Assessment
Pressure Ulcers/Pressure injuries (Braden scale)
CT & MR Angiography
CT & MR Angiography
Nursing Care and Pathophysiology for Diabetes Insipidus (DI)
Nursing Care and Pathophysiology for Disseminated Intravascular Coagulation (DIC)
Nursing Care and Pathophysiology of Glomerulonephritis
Isotonic Solutions (IV solutions)
Nursing Care and Pathophysiology of Osteoarthritis (OA)
Nursing Care and Pathophysiology for Pancreatitis
Preoperative (Preop) Education
Cerebral Angiography
Cerebral Angiography
Cerebral Angiography
Hypotonic Solutions (IV solutions)
Nursing Care and Pathophysiology of Osteoporosis
Nursing Care and Pathophysiology for Peptic Ulcer Disease (PUD)
Preoperative (Preop) Nursing Priorities
Thrombocytopenia
Blood Transfusions (Administration)
Cardiovascular Angiography
Cardiovascular Angiography
Cardiovascular Angiography
Fractures
Nursing Care and Pathophysiology for Hyperthyroidism
Hypertonic Solutions (IV solutions)
Integumentary (Skin) Important Points
Preload and Afterload
Nursing Care and Pathophysiology of Urinary Tract Infection (UTI)
Echocardiogram (Cardiac Echo)
Echocardiogram (Cardiac Echo)
Echocardiogram (Cardiac Echo)
Nursing Care and Pathophysiology for Hypothyroidism
Performing Cardiac (Heart) Monitoring
Ultrasound
Ultrasound
Interventional Radiology
Interventional Radiology
Nuclear Medicine
Cardiac Stress Test
Cardiac Stress Test
Pulmonary Function Test
Pulmonary Function Test
Endoscopy & EGD
Endoscopy & EGD
Colonoscopy
Colonoscopy
Mammogram
Biopsy
Biopsy
Electroencephalography (EEG)
Electroencephalography (EEG)
Electromyography (EMG)
Electromyography (EMG)
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology for Appendicitis
Nursing Care and Pathophysiology of Chronic Kidney (Renal) Disease (CKD)
Nursing Care and Pathophysiology of Diabetes Mellitus (DM)
General Anesthesia
Leukemia
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Diabetes Management
Dialysis & Other Renal Points
Local Anesthesia
Lymphoma
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Chloride-Cl (Hyperchloremia, Hypochloremia)
Nursing Care and Pathophysiology of Diabetic Ketoacidosis (DKA)
Moderate Sedation
Oncology Important Points
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Hyperglycaemic Hyperosmolar Non-ketotic syndrome (HHNS)
Nursing Care and Pathophysiology for Inflammatory Bowel Disease (IBD)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Malignant Hyperthermia
Phosphorus-Phos
Nursing Care and Pathophysiology for Ulcerative Colitis(UC)
Nursing Care and Pathophysiology for Crohn’s Disease
Normal Sinus Rhythm
Post-Anesthesia Recovery
Nursing Care and Pathophysiology for Acquired Immune Deficiency Syndrome (AIDS)
Nursing Care and Pathophysiology for Cholecystitis
Nursing Care and Pathophysiology for Heart Failure (CHF)
Postoperative (Postop) Complications
Sinus Bradycardia
Nursing Care and Pathophysiology for Anaphylaxis
Nursing Care and Pathophysiology for Hepatitis (Liver Disease)
Sinus Tachycardia
Nursing Care and Pathophysiology for Cirrhosis (Liver Disease, Hepatic encephalopathy, Portal Hypertension, Esophageal Varices)
Discharge (DC) Teaching After Surgery
Pacemakers
Atrial Fibrillation (A Fib)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Nursing Care and Pathophysiology for Pelvic Inflammatory Disease (PID)
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology for Endometriosis
Nursing Care and Pathophysiology for Menopause
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Absolute Neutrophil Count (ANC) Lab Values
Absolute Reticulocyte Count (ARC) Lab Values
Alanine Aminotransferase (ALT) Lab Values
Albumin Lab Values
Alkaline Phosphatase (ALK PHOS) Lab Values
Alpha-fetoprotein (AFP) Lab Values
Ammonia (NH3) Lab Values
Anion Gap
Antinuclear Antibody Lab Values
Base Excess & Deficit
Beta Hydroxy (BHB) Lab Values
Bicarbonate (HCO3) Lab Values
Blood Urea Nitrogen (BUN) Lab Values
Brain Natriuretic Peptide (BNP) Lab Values
C-Reactive Protein (CRP) Lab Values
Carbon Dioxide (Co2) Lab Values
Carboxyhemoglobin Lab Values
Cardiac (Heart) Enzymes
Cholesterol (Chol) Lab Values
Coagulation Studies (PT, PTT, INR)
Congestive Heart Failure (CHF) Labs
COPD (Chronic Obstructive Pulmonary Disease) Labs
Cortisol Lab Vales
Creatine Phosphokinase (CPK) Lab Values
Creatinine (Cr) Lab Values
Creatinine Clearance Lab Values
Cultures
Cyclic Citrullinated Peptide (CCP) Lab Values
D-Dimer (DDI) Lab Values
Direct Bilirubin (Conjugated) Lab Values
Dysrhythmias Labs
Erythrocyte Sedimentation Rate (ESR) Lab Values
Fibrin Degradation Products (FDP) Lab Values
Fibrinogen Lab Values
Fluid Compartments
Free T4 (Thyroxine) Lab Values
Gamma Glutamyl Transferase (GGT) Lab Values
Glomerular Filtration Rate (GFR)
Glucagon Lab Values
Glucose Lab Values
Glucose Tolerance Test (GTT) Lab Values
Growth Hormone (GH) Lab Values
Hematocrit (Hct) Lab Values
Hemodynamics
Hemoglobin (Hbg) Lab Values
Hemoglobin A1c (HbA1C)
Hepatitis B Virus (HBV) Lab Values
Homocysteine (HCY) Lab Values
Ionized Calcium Lab Values
Iron (Fe) Lab Values
Ischemic (CVA) Stroke Labs
Lab Panels
Lab Values Course Introduction
Lactate Dehydrogenase (LDH) Lab Values
Lactic Acid
Lipase Lab Values
Lithium Lab Values
Liver Function Tests
Mean Corpuscular Volume (MCV) Lab Values
Mean Platelet Volume (MPV) Lab Values
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
Methemoglobin (MHGB) Lab Values
Myoglobin (MB) Lab Values
Order of Lab Draws
Pediatric Bronchiolitis Labs
Phosphorus (PO4) Blood Test Lab Values
Platelets (PLT) Lab Values
Pneumonia Labs
Potassium-K (Hyperkalemia, Hypokalemia)
Prealbumin (PAB) Lab Values
Pregnancy Labs
Procalcitonin (PCT) Lab Values
Prostate Specific Antigen (PSA) Lab Values
Protein (PROT) Lab Values
Protein in Urine Lab Values
Red Blood Cell (RBC) Lab Values
Red Cell Distribution Width (RDW) Lab Values
Renal (Kidney) Failure Labs
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Sepsis Labs
Shorthand Lab Values
Nursing Care and Pathophysiology for SIADH (Syndrome of Inappropriate antidiuretic Hormone Secretion)
Thyroid Stimulating Hormone (TSH) Lab Values
Thyroxine (T4) Lab Values
Total Bilirubin (T. Billi) Lab Values
Total Iron Binding Capacity (TIBC) Lab Values
Triiodothyronine (T3) Lab Values
Troponin I (cTNL) Lab Values
Urinalysis (UA)
Urine Culture and Sensitivity Lab Values
Vitamin B12 Lab Values
Vitamin D Lab Values
White Blood Cell (WBC) Lab Values