Nursing Care and Pathophysiology for Cardiogenic Shock

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Nichole Weaver
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Included In This Lesson

Study Tools For Nursing Care and Pathophysiology for Cardiogenic Shock

Shock – Signs and symptoms (Mnemonic)
Cardiogenic Shock Pathochart (Cheatsheet)
Shock (Cheatsheet)
Dopamine vs Dobutamine (Cheatsheet)
Pulmonary Artery Catheter (Image)
Cardiac Tamponade (Image)
Types of Shock (Picmonic)
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Outline

  1. Causes
    1. Myocardial infarction (MI)
    2. End-stage cardiomyopathy
    3. Papillary muscle or valve rupture
    4. Cardiac tamponade
    5. Pulmonary embolism (PE)
    6. Sudden, severe, extreme heart failure
  2.  Decreased Perfusion
    1. ↓ CO
    2. ↓ BP
    3. ↑ HR (compensation)
    4. ↑ SVR (compensation)
    5. Weak, thready pulses
    6. Cool, diaphoretic skin
    7. Pale, dusky, cyanotic, or mottled skin
    8. ↓ urine output
    9. ↓ LOC, anxiety
    10. Weakness
  3.  Volume Overload
    1. ↑ CVP
    2. JVD
    3. Pulmonary Edema
    4. Crackles
    5. Pink, frothy sputum
    6. Sudden, severe SOB
    7. Muffled Heart Sounds
    8. S3, S4 present
  4.  Therapeutic Management
    1. Treat Cause
      1. Revascularization for MI (PCI, CABG)
      2. Thrombolytics or surgical removal for PE
      3. Pericardiocentesis for cardiac tamponade
    2. Improve Contractility
      1. Dopamine – may ↑ HR
      2. Dobutamine
    3. Decrease Afterload
      1. Dobutamine
      2. Diuretics
      3. Furosemide – for Pulmonary edema
        1. Caution – may ↓ BP
    4. Surgical Intervention
      1. IABP
      2. LVAD
      3. Heart Transplant
  5.  Patient Education
    1. Health promotion for prevention of myocardial infarction (see MI lesson)
    2. Explain procedures and expectations
    3. Symptoms to report to RN or HCP
    4. Importance of positioning (HOB > 30° and legs elevated)
    5. Medication instructions, side effects

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Transcript

Okay guys, let’s talk about the basics of cardiogenic shock.

Remember the initial insult here is a broken or blocked pump. There are three main causes – myocardial infarction (remember dead heart muscle can’t pump), cardiac tamponade (which is fluid around the heart that compresses it and makes it hard to pump), and a pulmonary embolism that keeps blood from moving forward through the lungs. So the big thing we want you to see is that this is like an extreme version of heart failure. The blood can’t go forward so it backs up – so we have an increased preload. The bad pump causes our cardiac output and blood pressure to drop. The body tries to compensate by increasing the heart rate, and also by vasoconstricting and shunting blood to vital organs, so we see our afterload increase as well. What we’re left with is a pumping system that is entirely broken and vital organs that aren’t getting the blood they need. So what does this look like in our patient?

Well remember it’s like extreme, sudden, severe heart failure – so you’ve lost all perfusion forward. What happens with a lack of perfusion to the brain? Decreased LOC. Decreased perfusion to the kidneys? Decreased urine output. Decreased perfusion to the skin and extremities? Cold, pale, clammy skin, decreased pulses, slow cap refill. ALL signs of decreased perfusion. That’s your #1 clue. We may also see the problems of the blood backing up, right? Remember what happens when the blood backs up into the lungs – we get pulmonary edema and hear crackles. Then when it starts backing up into the head we see that extreme JVD – like a rope in their neck, right? So can you see how this is just sudden, extreme, severe heart failure? So keep that in mind as we move forward.

When we start thinking about treating a patient in cardiogenic shock, the most important thing is to identify and treat the cause. If they’ve had an MI, they’ll need revascularization like we talked about in the MI lesson. If it’s cardiac tamponade we need to remove the fluid from around their heart, if it’s a pulmonary embolism, we need to bust up or remove that clot. If we don’t address the cause, nothing else we do will be effective. When it comes to medication management, the big goal is to get the heart pumping more effectively and decrease the pressure it has to pump against. The top two drugs we give for patients in cardiogenic shock are dopamine and dobutamine. They will both increase contractility. Dopamine can also increase heart rate, while dobutamine can also help with vasodilation to decrease afterload. Patients who have had an MI will also still get the standard MONA treatment as well. If the patient has developed severe pulmonary edema, they may also receive a diuretic like Furosemide to offload that volume. We just have to be careful not to drop their blood pressure too much.

So, there are a lot of nursing priorities for this patient – one of which being that they need to be in an ICU. But we’re gonna focus on the top 3 concepts here. If you check out the outline and the care plan attached to this lesson, you’ll see a ton of details about specific interventions. The first concept is perfusion, we have got to monitor their hemodynamics and maintain a good cardiac output. Then, because these patients are at risk for airway and breathing issues, we need to monitor their oxygen status and intervene as needed. Then finally I added clotting because this condition might be caused by a clot in the coronary arteries or pulmonary arteries or might be because of bleeding around the heart – so we need to consider the interventions required to manage those conditions as well.

So let’s recap – cardiogenic shock is caused by a broken or blocked pump – that might be an MI, cardiac tamponade, or a PE. Most of the symptoms you see are caused by a lack of perfusion to the organs like the brain, kidneys, and skin, and by the backup of blood into the lungs and body (that’s why you see pulmonary edema and JVD). Remember it’s like a sudden, extreme version of heart failure. Treatment is focused on treating the cause, increasing contractility and getting the heart pumping more efficiently against less pressure. Our priorities are going to be perfusion and oxygenation, and then dealing with any clotting or bleeding issues depending on the cause. And finally, remember that this is an emergency, these patients need to be in an ICU and may even need to be on life support. So don’t be afraid to ask for help if you need it!

Make sure you check out the care plan and outline in this lesson to see lots of details about nursing care and interventions. We love you guys! Happy nursing!

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NP 4 Exam 2

Concepts Covered:

  • Circulatory System
  • Urinary System
  • Adult
  • Basic
  • Test Taking Strategies
  • Prefixes
  • Suffixes
  • Integumentary Disorders
  • Respiratory Disorders
  • Pediatric
  • Bipolar Disorders
  • Immunological Disorders
  • Labor Complications
  • Neonatal
  • Medication Administration
  • Disorders of Pancreas
  • Pregnancy Risks
  • Cardiac Disorders
  • Learning Pharmacology
  • Eating Disorders
  • Dosage Calculations
  • Emergency Care of the Cardiac Patient
  • Substance Abuse Disorders
  • Vascular Disorders
  • Endocrine and Metabolic Disorders
  • Shock
  • Fetal Development
  • Depressive Disorders
  • Anxiety Disorders
  • Cardiovascular Disorders
  • Liver & Gallbladder Disorders
  • Upper GI Disorders
  • Female Reproductive Disorders
  • Neurologic and Cognitive Disorders
  • Personality Disorders
  • Nervous System
  • Urinary Disorders
  • Hematologic Disorders
  • Disorders of the Posterior Pituitary Gland
  • Respiratory System
  • Renal Disorders
  • Noninfectious Respiratory Disorder
  • Shock

Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Life Support Review Course Introduction
12 Points to Answering Pharmacology Questions
CPR-BLS (Basic Life Support)
Electrical A&P of the Heart
54 Common Medication Prefixes and Suffixes
Advanced Cardiovascular Life Support (ACLS)
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Vitals (VS) and Assessment
Fluid Shifts (Ascites) (Pleural Effusion)
Pediatric Advanced Life Support (PALS)
Therapeutic Drug Levels (Digoxin, Lithium, Theophylline, Phenytoin)
Essential NCLEX Meds by Class
Isotonic Solutions (IV solutions)
Neonatal Resuscitation Program (NRP)
6 Rights of Medication Administration
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
The SOCK Method – Overview
The SOCK Method – S
The SOCK Method – O
The SOCK Method – C
The SOCK Method – K
Basics of Calculations
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Dimensional Analysis Nursing (Dosage Calculations/Med Math)
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Oral Medications
Chloride-Cl (Hyperchloremia, Hypochloremia)
Injectable Medications
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
IV Infusions (Solutions)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Complex Calculations (Dosage Calculations/Med Math)
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Nursing Care and Pathophysiology for Heart Failure (CHF)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Atrial Flutter
Pacemakers
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Benzodiazepines
Nursing Care and Pathophysiology of Hypertension (HTN)
Cardiac (Heart) Disease in Pregnancy
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Dehydration
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Fetal Circulation
MAOIs
SSRIs
TCAs
Congenital Heart Defects (CHD)
Defects of Increased Pulmonary Blood Flow
Defects of Decreased Pulmonary Blood Flow
Insulin
Obstructive Heart (Cardiac) Defects
Mixed (Cardiac) Heart Defects
Histamine 1 Receptor Blockers
Histamine 2 Receptor Blockers
Renin Angiotensin Aldosterone System
ACE (angiotensin-converting enzyme) Inhibitors
Angiotensin Receptor Blockers
Calcium Channel Blockers
Cardiac Glycosides
Metronidazole (Flagyl) Nursing Considerations
Ciprofloxacin (Cipro) Nursing Considerations
Vancomycin (Vancocin) Nursing Considerations
Anti-Infective – Penicillins and Cephalosporins
Atypical Antipsychotics
Autonomic Nervous System (ANS)
Sympathomimetics (Alpha (Clonodine) & Beta (Albuterol) Agonists)
Parasympathomimetics (Cholinergics) Nursing Considerations
Parasympatholytics (Anticholinergics) Nursing Considerations
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Epoetin Alfa
HMG-CoA Reductase Inhibitors (Statins)
Magnesium Sulfate
NSAIDs
Corticosteroids
Hydralazine (Apresoline) Nursing Considerations
Nitro Compounds
Vasopressin
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Base Excess & Deficit
Blood Flow Through The Heart
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Coronary Circulation
Fluid Compartments
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
Lactic Acid
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Pacemakers
Performing Cardiac (Heart) Monitoring
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Proton Pump Inhibitors
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Shock Module Intro
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)