EKG (ECG) Waveforms

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Included In This Lesson

Study Tools For EKG (ECG) Waveforms

Parts of EKG waveform (Image)
EKG Chart (Cheatsheet)
EKG Electrical Activity Worksheet (Cheatsheet)
10 Common EKG Heart Rhythms (Cheatsheet)
ECG Interpretation (Picmonic)
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Outline

Overview

  1. The heart’s electrical activity that stimulates the atria and ventricles to contract produce a waveform on an EKG
  2. These waveforms are broken down into in a P wave, QRS complex and T wave.

 

Nursing Points

  1. P wave
    1. Atrial  depolarization 
      1. Positive deflection 
    2. PR interval 
      1. Beginning of P wave to beginning of QRS
      2. Time it takes for electrical current  to reach ventricles
        1. 0.12-0.20 seconds
  2. QRS Complex
    1. Ventricular depolarization
      1. Negative and positive deflection
    2. QRS interval
      1. Beginning of the Q wave to ending of S wave
      2. Time it takes for the electrical current to travel through the ventricles
        1. 0.06 -012 seconds
  3. T wave
    1. Ventricular repolarization
      1. Positive deflection
    2. QT interval
      1. Beginning of QRS to the end of T wave
      2. Time it takes for the ventricles to contract and relax
        1. 0.36-.044 seconds
    3. ST segment 
      1. End of QRS complex to the beginning of T wave
      2. Time between ventricular depolarization and repolarization

Assessment

  1. Recognize PQRST waveforms on EKG
  2. Check pulse if abnormal waveforms are observed

 

Therapeutic Management

  1. Recognize and report abnormal waveforms 
    1. Long PR interval
    2. Prolonged  QRS Complex
    3. Tall T waves

 

Nursing Concepts

  1. EKG Rhythms

 

 

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Transcript

Hey guys in this lesson we are going to talk about the EKG waveforms. As nurses, it is imperative to have a basic understanding of these waveforms so we can quickly identify abnormalities in our patient’s heart rhythms.

So let’s get started! Every single time the heart contracts and we have a heartbeat there is an electrical waveform that is created on an EKG. Remember from our other lessons that when there is an electrical conduction it causes a mechanical action. So every time the atria contract it produces a P wave and when the ventricles contract it produces a QRS. And when the ventricles relax or depolarize, a T wave is produced. Let’s break down each of these waveforms into more detail!

Let’s start with the P wave. Remember the SA node, every single time the SA node sends an electrical impulse across the atria, the atria contract or depolarize. When this action takes place, it creates the P wave on an EKG, the P wave is an upward or positive deflection. The PR interval on an EKG is very important also, the PR interval is measured from the beginning of the P wave to the beginning of the QRS. This PR interval indicates the seconds it takes for the electrical current to reach the ventricles. When SA node starts the electrical impulse and the atria contract, the impulse is then sent to the AV node, remember that the AV node slows down the impulse so the atria can contract all the way. Then the AV node and bundle of His send the impulse to the ventricles. So the time it takes for the atria to initiate a contraction, contract and send the impulse to the ventricles is called a PR interval. This is normally done in 0.12-0.20 seconds. That’s not a lot of time! Anything longer than that, like 0.24-0.28 seconds means that it is taking the electrical current too long to get to the ventricles. That is when people have first degree heart blocks but we will talk about those in another lesson!

So after the atria has finished contracting, it sends the impulse to the ventricles. When the ventricles contract or depolarize, a QRS complex is seen on an EKG. This waveform has negative and positive waveforms or downward and upward waveforms depending on the lead. As you can see the waveform on a QRS complex, especially the R wave is a higher amplitude, that is because of the force of contraction the ventricle has to pump to effectively get the blood out to the body creates a greater voltage and amplitude. The QRS interval is the time it takes for the electrical current to travel through the ventricles, it starts at the beginning of the Q wave to the end of the S wave. It should normally take about 0.06-0.12 seconds to complete. Again that is not that long, the heart should not take that long to contract. The heart naturally beats about 80 times a minute so it needs to do this pretty quickly to keep up. If a QRS interval is long, you will hear the term wide QRS complex and it means that its usually greater than 0.12 seconds and its taking the heart way too long to contract.

And lastly, the good old T wave. It is the last waveform on an EKG. When the electrical conduction has finished and depolarization is complete, the ventricles repolarize so they can rest and fill up with blood and get ready for the next contraction. When potassium is reentering the cell, it causes the ventricle to relax, this is what produces the T wave on an EKG. This is a positive deflection. The QT interval on an EKG is the time it takes for the ventricles to contract and relax, it starts at the beginning of the Q wave and ends at the end of the T wave, it should normally take 0.36-0.44 seconds for this to take place. Your might hear prolonged QT interval also and this means that it takes longer than 0.44 seconds for the ventricle to contract and relax, this is not good. When the ventricles take too long to contract and relax, it can cause deadly arrhythmias. So always keep an eye out for that QT interval. ST segment is from the end of the QRS complex to the beginning of the T wave, it’s a flat line and it’s the time between ventricular depolarization and repolarization.

It is important to understand that the PQRST waveforms can indicate what is going on in the heart and which chamber is contracting and when. Meaning, when you see a P wave you know that is the atria contracting, if there are abnormal P waves, you would know it has to do with the atria. Same with the QRS, it means the ventricles are contracting, so when you see abnormalities in the QRS complex, you will know it has something to do with the ventricles. Of course the T wave is the ventricles relaxing, so if the T wave is abnormal you would know the ventricles are not relaxing like they should. With that being said, it is also important to understand that the atria contract and then the ventricles contract. It just simply has to work that way, therefore you should always see a P wave followed by a QRS. This is how you can always tell if you patient is a sinus rhythm. Do you have a 1 P wave followed by 1 QRS, and of course a T wave. Sometimes there is a U wave which is not clearly understood but it’s thought to be repolarization of the Purkinje fibers. Not everyone has a U wave so you may not see them too often.

All right so you guys. It is so important, I don’t think I can stress this enough how important it is for you to understand that pqrst wave form because it helps you identify which chamber is contrasting and at what time.
So again looking at this, we have a P wave, P wave. P waves tell, if you recall every time you see your P wave it means what the Atria is contracting or is depolarizing. And then we identify our QRS, QRS, QRS complex. Every time that we see a QRS, it means what guys? The ventricles are contracting. So you should always always always see a P wave followed by a QRS, P wave followed by a QRS, P wave followed by a QRS. And then of course don’t forget about our T waves. T waves are very important, it means that the ventricles are relaxing. So you might ask yourself, how come you don’t have a waveform for the Atria to relax? Well that usually happens around here. So because the qrs is so much bigger, you don’t see a waveform where the Atria are relaxing. So with this being said, you know that the P wave means the Atria are Contracting. So you should always see a P wave followed by a QRS because the Atria contract and the ventricles contract, atria contracts, ventricles contract, so you should always have a p wave followed by a QRS. And of course if the ventricles contract they have to relax. If not, we have a dead patient. So you always have to see a t wave. Every now and then, I’m not sure if you guys have ever heard of a u wave, every now and then a patient may have a u wave, and this is usually occurs right after the T wave. It’s not really understood what they u wave is there for it they believe that it may be repolarization of the purkinje fibers. But not everybody has one. In my career I think I’ve seen like two patients with a u wave but what’s interesting about that is that you may get it confused and think it’s a p wave. Cuz there’s a T waves pqrst and then a u wave, the you’ll see a P wave again followed by a PQRST. So here’s our new but I still sometimes when you look at it you’re like wait a minute or is that an extra P wave what is it? And then you get everybody involved and you start analyzing it until you realize no, this patient has a U-wave. So again, it doesn’t happen too often and you will not see it that often. But just know that every now and then very rarely a u wave decides to come along whenever it feels like it. That just depends on the patient, their electrical conduction, on whether it shows up.
To recap, these key points are your biggest takeaway from this lesson. Remember to keep it simple so you can understand the basics. The P wave is when the atria depolarize, the PR interval is how long it takes for the electrical current to reach the ventricles. The QRS complex is ventricular depolarization and the QRS interval is how long it takes for the electrical current to travel through the ventricles and the T wave is when the ventricles repolarize.

I hope this makes sense to you guys and that you are able to understand the basic waveforms on an EKG. Make sure you check out all of the resources attached to this lesson. Now, go out and be your best self today! And, as always, happy nursing!

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Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Life Support Review Course Introduction
12 Points to Answering Pharmacology Questions
CPR-BLS (Basic Life Support)
Electrical A&P of the Heart
54 Common Medication Prefixes and Suffixes
Advanced Cardiovascular Life Support (ACLS)
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Vitals (VS) and Assessment
Fluid Shifts (Ascites) (Pleural Effusion)
Pediatric Advanced Life Support (PALS)
Therapeutic Drug Levels (Digoxin, Lithium, Theophylline, Phenytoin)
Essential NCLEX Meds by Class
Isotonic Solutions (IV solutions)
Neonatal Resuscitation Program (NRP)
6 Rights of Medication Administration
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
The SOCK Method – Overview
The SOCK Method – S
The SOCK Method – O
The SOCK Method – C
The SOCK Method – K
Basics of Calculations
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Dimensional Analysis Nursing (Dosage Calculations/Med Math)
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Oral Medications
Chloride-Cl (Hyperchloremia, Hypochloremia)
Injectable Medications
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
IV Infusions (Solutions)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Complex Calculations (Dosage Calculations/Med Math)
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Nursing Care and Pathophysiology for Heart Failure (CHF)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Atrial Flutter
Pacemakers
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Benzodiazepines
Nursing Care and Pathophysiology of Hypertension (HTN)
Cardiac (Heart) Disease in Pregnancy
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Dehydration
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Fetal Circulation
MAOIs
SSRIs
TCAs
Congenital Heart Defects (CHD)
Defects of Increased Pulmonary Blood Flow
Defects of Decreased Pulmonary Blood Flow
Insulin
Obstructive Heart (Cardiac) Defects
Mixed (Cardiac) Heart Defects
Histamine 1 Receptor Blockers
Histamine 2 Receptor Blockers
Renin Angiotensin Aldosterone System
ACE (angiotensin-converting enzyme) Inhibitors
Angiotensin Receptor Blockers
Calcium Channel Blockers
Cardiac Glycosides
Metronidazole (Flagyl) Nursing Considerations
Ciprofloxacin (Cipro) Nursing Considerations
Vancomycin (Vancocin) Nursing Considerations
Anti-Infective – Penicillins and Cephalosporins
Atypical Antipsychotics
Autonomic Nervous System (ANS)
Sympathomimetics (Alpha (Clonodine) & Beta (Albuterol) Agonists)
Parasympathomimetics (Cholinergics) Nursing Considerations
Parasympatholytics (Anticholinergics) Nursing Considerations
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Epoetin Alfa
HMG-CoA Reductase Inhibitors (Statins)
Magnesium Sulfate
NSAIDs
Corticosteroids
Hydralazine (Apresoline) Nursing Considerations
Nitro Compounds
Vasopressin
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Base Excess & Deficit
Blood Flow Through The Heart
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Coronary Circulation
Fluid Compartments
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
Lactic Acid
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Pacemakers
Performing Cardiac (Heart) Monitoring
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Proton Pump Inhibitors
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Shock Module Intro
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)