Ventricular Tachycardia (V-tach)

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Maria Stewart
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Included In This Lesson

Study Tools For Ventricular Tachycardia (V-tach)

Parts of EKG waveform (Image)
Ventricular Tachycardia (Image)
10 Common EKG Heart Rhythms (Cheatsheet)
EKG Chart (Cheatsheet)
EKG Electrical Activity Worksheet (Cheatsheet)
Heart Rhythms Signs and Symptoms (Cheatsheet)
Heart Rhythm Identification (Cheatsheet)
Hs and Ts of ACLS (Cheatsheet)
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Outline

Overview

  1. Ventricular Tachycardia
    1. Multiple unorganized electrical signals in the ventricles
      1. Ventricles contract at a rate of 150-250 bpm
      2. May or may not have pulse
      3. Significantly reduces CO and perfusion

Nursing Points

General

  1. Characteristics of Ventricular tachycardia
    1. Rhythm
      1. Regular
      2. Irregular
    2. Rate
      1. 150-250 bpm
        1. Ventricular rate
    3. P:QRS ratio
      1. No  P waves
        1. Not measurable
    4. PR interval
      1. No P waves
        1. Not measurable
    5. QRS complex
      1. > 0.12 seconds  
      2. “Wide”

Assessment

  1. Patient Presentation
    1. Palpitations
    2. Chest pain
    3. Decreased CO
      1. Hypotensive
      2. LOC changes
      3. Lightheaded
      4. Syncope
  2. Pulse or pulseless
  3. Electrolytes

Therapeutic Management

  1. Nursing Interventions
    1. Determine if a pulse is present
    2. Sustained or Unsustained
      1. Monomorphic
      2. Polymorphic
  2. Determine/Treat the cause
    1. Electrolytes
    2. MI
    3. Abnormal heart conditions
  3. Follow ACLS guidelines
    1. V-tach with pulse
      1. Amiodarone IV
      2. Magnesium Sulfate IV
      3. Synchronized Cardioversion (CV)
    2. Pulseless V-tach
      1. CPR
      2. Defibrillate
      3. Epinephrine

Nursing Concepts

  1. EKG Rhythms
  2. Perfusion

Patient Education

  1. Seek medical help

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Transcript

Hey guys, so in this lesson we are going to talk about ventricular tachycardia, also called V-tach. We are going to break down the characteristics of it on an EKG and talk about nursing interventions and treatments for V-tach. It is a pretty important rhythm to recognize because people can die quickly if we don’t do something about it. So let’s get started!

So in Ventricular Tachycardia there are multiple unorganized electrical signals in the ventricles, this causes the ventricles to contract at a rate of 150-250 beats per minute. Because the ventricles do not slow down enough to fill back up with blood, cardiac output is significantly decreased. Because of that, a person who is in V-tach may or may not have a pulse so it is imperative to assess that when you see this rhythm.

So let’s use the 6-step method and break down the characteristics of V-tach. So in step 1 we need to determine if the rhythm is regular or irregular, so if need to try to find the number of boxes in between the R waves so we will try from here to here and we have about 11-12 and here to here 10-11 and here to here 12. So our rhythm is regular with V-tach it will be regular or irregular. in step 2 let’s count the heart rate, we multiply 13 by 10 and get 130. But let’s also do the 1500 method for a more accurate rate, so 1500 divided by 11 and we get 136 beats per minute. In step 3 we need to look at the P:QRS ratio, and that is not measurable since we do not have P waves. In step 4 we look at the PR Interval and again it is not measurable since we do not have P waves. In step 5 we look at the QRS complex so if we measure from here to here we have 11 small boxes or 0.44 seconds. So in step 6 we identify our rhythm and it is ventricular tachycardia. Let’s recap the characteristics, the rhythm can be regular or irregular, the rate is between 150-250 beats per minute for the most part. The P:QRS ratio and PR interval is not measurable since there are no P waves. The QRS complex is wide. This is one of those rhythms that when you see it you don’t not stop and count anything or go through the steps, you should recognize it right away and go check on your patient! This is not one of those rhythms you need to analyze, this is one of those rhythms that make you jump! People can go into cardiac arrest and die if we don’t do anything about this rhythm. So now let’s talk about managing V-tach.

So most patients that come in with Ventricular tachycardia will have palpitations, chest pain and because of the decreased cardiac output, they will be hypotensive, with altered level of consciousness, they will also be lightheaded have syncope. The priority nursing interventions are to find out if they have a pulse or not the second you see this rhythm. People with v-tach can or cannot have a pulse, it depends on the severity of the cardiac output. Find out if it is sustained or unsustained, if there is a patient in a normal sinus rhythm and all of a sudden they have a run of v-tach and go back to normal sinus rhythm, we call this unsustained and it is just a run of vtach. If it is sustained, it’s not going away. You also need to find out if it is monomorphic v-tach or polymorphic v-tach. Basically if all of the QRS look the same it is monomorphic if they look different it’s polymorphic like in this strip here. This would be called polymorphic ventricular tachycardia.

So therapeutic management for ventricular tachycardia is to determine the cause and treat it, it may be something so simple as an electrolyte abnormality or an MI. We also need to follow the ACLS guidelines for V-tach. So once you determine if the patient has a pulse then we treat it with Amiodarone 150 mg IV or 1 or 2 grams of Mag Sulfate. We can also do a synchronized cardioversion if the meds do not work and the V-tach persists. Now if they are unstable and do not have a pulse, we treat that differently. They don’t have a pulse so there is no cardiac output, they are going to die! So we need to start CPR immediately and defibrillate them and give epinephrine according to the ACLS guidelines. A quick note, when you hear synchronized or unsynchronized cardioversion, basically when the defibrillator machine is set to synchronize it synch with the R waves to deliver a low voltage shock after repolarization. When you hear unsynchronized it is the same as defibrillate and it means a higher voltage shock is delivered as soon as the button is pushed. Make sure no one is touching the patient!

So the key points to take a way from this lesson are to remember the abnormalities of ventricular tachycardia. The ventricles are rapidly contracting at a rate of 150-250 beats per minute. People may or may not have a pulse with V-tach. So the priority nursing intervention is to assess the patient first and see if there is a pulse present. Then follow the ACLS guidelines, so if there is a pulse we try medications first then a cardioversion, if there is not a pulse we need to do CPR and defibrillate them and give them meds to save their life.

I hope that you guys have enjoyed this lesson and feel more comfortable identifying ventricular tachycardia and know what interventions to implement. Make sure you check out all of the resources attached to this lesson. Now, go out and be your best self today! And, as always, happy nursing!

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Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Life Support Review Course Introduction
12 Points to Answering Pharmacology Questions
CPR-BLS (Basic Life Support)
Electrical A&P of the Heart
54 Common Medication Prefixes and Suffixes
Advanced Cardiovascular Life Support (ACLS)
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Vitals (VS) and Assessment
Fluid Shifts (Ascites) (Pleural Effusion)
Pediatric Advanced Life Support (PALS)
Therapeutic Drug Levels (Digoxin, Lithium, Theophylline, Phenytoin)
Essential NCLEX Meds by Class
Isotonic Solutions (IV solutions)
Neonatal Resuscitation Program (NRP)
6 Rights of Medication Administration
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
The SOCK Method – Overview
The SOCK Method – S
The SOCK Method – O
The SOCK Method – C
The SOCK Method – K
Basics of Calculations
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Dimensional Analysis Nursing (Dosage Calculations/Med Math)
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Oral Medications
Chloride-Cl (Hyperchloremia, Hypochloremia)
Injectable Medications
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
IV Infusions (Solutions)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Complex Calculations (Dosage Calculations/Med Math)
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Nursing Care and Pathophysiology for Heart Failure (CHF)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Atrial Flutter
Pacemakers
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Benzodiazepines
Nursing Care and Pathophysiology of Hypertension (HTN)
Cardiac (Heart) Disease in Pregnancy
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Dehydration
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Fetal Circulation
MAOIs
SSRIs
TCAs
Congenital Heart Defects (CHD)
Defects of Increased Pulmonary Blood Flow
Defects of Decreased Pulmonary Blood Flow
Insulin
Obstructive Heart (Cardiac) Defects
Mixed (Cardiac) Heart Defects
Histamine 1 Receptor Blockers
Histamine 2 Receptor Blockers
Renin Angiotensin Aldosterone System
ACE (angiotensin-converting enzyme) Inhibitors
Angiotensin Receptor Blockers
Calcium Channel Blockers
Cardiac Glycosides
Metronidazole (Flagyl) Nursing Considerations
Ciprofloxacin (Cipro) Nursing Considerations
Vancomycin (Vancocin) Nursing Considerations
Anti-Infective – Penicillins and Cephalosporins
Atypical Antipsychotics
Autonomic Nervous System (ANS)
Sympathomimetics (Alpha (Clonodine) & Beta (Albuterol) Agonists)
Parasympathomimetics (Cholinergics) Nursing Considerations
Parasympatholytics (Anticholinergics) Nursing Considerations
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Epoetin Alfa
HMG-CoA Reductase Inhibitors (Statins)
Magnesium Sulfate
NSAIDs
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Hydralazine (Apresoline) Nursing Considerations
Nitro Compounds
Vasopressin
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Base Excess & Deficit
Blood Flow Through The Heart
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Coronary Circulation
Fluid Compartments
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
Lactic Acid
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Pacemakers
Performing Cardiac (Heart) Monitoring
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Proton Pump Inhibitors
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Shock Module Intro
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)