Coronary Circulation

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Jon Haws
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Included In This Lesson

Study Tools For Coronary Circulation

Coronary Arteries – Location (Mnemonic)
Coronary Circulation (Cheatsheet)
Coronary Anatomy (Image)
Coronary Arteries (Picmonic)
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Outline

Overview

Heart has its own system of arteries to supply blood to myocardium

Nursing Points

General

  1. Coronary circulation branches off aorta above aortic valve
  2. Coronary perfusion during diastole
  3. Aorta
    1. Right Coronary Artery (RCA)
      1. Right Posterior Descending
      2. Right Marginal Artery
      3. SUPPLIES:
        1. RA
        2. RV
        3. Septum
        4. SA node
        5. AV node
    2. Left Main Coronary Artery (LMCA)
      1. Left Anterior Descending (LAD)
      2. Left Circumflex (LCX)
      3. SUPPLIES:
        1. LA
        2. LV
        3. Septum

Assessment

  1. Tachycardia
    1. Decreased diastolic time
    2. Decreased coronary perfusion
    3. High risk for ischemia / infarction

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Transcript

This lesson will be a quick review of coronary circulation – that is the the flow of blood to the heart muscle itself.
So, just as the heart is going to supply blood to the body, it also has to supply blood to itself. The heart is an enormous muscle and it requires a tremendous amount of cardiac output in order to just carry out its normal functions. So, because of that, the heart actually has its own system of arteries.

As you can see, the coronary arteries actually branch off of the aorta right above the aortic valve. That’s important to note, we’ll look at that in a second. On both sides of the heart you have a main coronary artery branching off – the Right Coronary Artery or RCA, and the Left Main Coronary Artery or LMCA. From there they each have smaller branches to supply the front and back of the heart. On the right we have the Right Posterior Descending which wraps around the back and the Right Marginal Artery which wraps around the front. On the left, we have the Left Anterior Descending or LAD that supplies the front left side of the heart and the Left Circumflex, often written LCX, which wraps around the back left side of the heart. You’ll notice there are more branches on the left, that’s because it is larger than the right. The main ones that are susceptible to blockage are the RCA, LMCA, LAD, and LCX – you’ll learn more about what a blockage means in the Myocardial Infarction lesson.

Now, it’s important to note what each of these coronary systems supplies blood to. On the left, it supplies the Left Atrium, Left Ventricle, and part of the Septum. On the right it supplies the Right Atrium, Right Ventricle, and part of the Septum as well, but it ALSO supplies the SA and AV node. So you can imagine that if someone had a blockage on the right side of their coronary perfusion, they might be susceptible to arrhythmias.

One more thing that’s important to note about how and when the heart perfuses itself. Remember how I said that the coronary circulation branches right above the aortic valve? Well if you think about systole and diastole. The heart contracts in systole and the Aortic Valve opens – when it does, it will briefly block the coronary circulation. Then the heart relaxes in diastole, the aortic valve closes, and blood flows through the coronary arteries. There is also a pause after diastole to allow filling of the ventricles, so this gives even more time for blood to flow to the coronary arteries. So, the heart perfuses the body during systole, and perfuses itself during diastole.

Time for some critical thinking! If my patient’s heart rate is extremely fast, what happens to their time in diastole? It decreases! Instead of Lub, Dub……..Lub, Dub…..Lub, Dub….. it becomes LubDubLubDubLubDub with no pauses in between. So if my heart perfuses itself during diastole, what happens to coronary perfusion in severe or prolonged tachycardia? It decreases dramatically!
It’s like pressing a water fountain button over and over really fast and expecting to be able to get a full drink of water. It’s not possible. You have to hold it. Without that diastolic pause, the heart struggles to perfuse itself.
In the ICU when we see a patient in a significant tachycardia (for example Supraventricular Tachycardia in the 160’s), who is stable so far, we all tend to say something like “can’t stay like that forever” or “his heart will give out eventually” because we know that the patient’s coronary perfusion is decreased significantly. So that’s just something I want you to know so that if you have a patient who’s been severely tachycardic for a long time, you will know how important it is to address the situation!
So remember, the coronary circulation is how the heart gets blood flow to itself. Both sides of the heart have a main coronary artery and then branches to cover the whole muscle. The heart perfuses the body during systole, but perfuses itself during diastole. And prolonged tachycardia can lead to decreased coronary perfusion and even a myocardial infarction.

I hope you learned something today! Go out and be your best self and as always, happy nursing!

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NP 4 Exam 2

Concepts Covered:

  • Circulatory System
  • Urinary System
  • Adult
  • Basic
  • Test Taking Strategies
  • Prefixes
  • Suffixes
  • Integumentary Disorders
  • Respiratory Disorders
  • Pediatric
  • Bipolar Disorders
  • Immunological Disorders
  • Labor Complications
  • Neonatal
  • Medication Administration
  • Disorders of Pancreas
  • Pregnancy Risks
  • Cardiac Disorders
  • Learning Pharmacology
  • Eating Disorders
  • Dosage Calculations
  • Emergency Care of the Cardiac Patient
  • Substance Abuse Disorders
  • Vascular Disorders
  • Endocrine and Metabolic Disorders
  • Shock
  • Fetal Development
  • Depressive Disorders
  • Anxiety Disorders
  • Cardiovascular Disorders
  • Liver & Gallbladder Disorders
  • Upper GI Disorders
  • Female Reproductive Disorders
  • Neurologic and Cognitive Disorders
  • Personality Disorders
  • Nervous System
  • Urinary Disorders
  • Hematologic Disorders
  • Disorders of the Posterior Pituitary Gland
  • Respiratory System
  • Renal Disorders
  • Noninfectious Respiratory Disorder
  • Shock

Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Life Support Review Course Introduction
12 Points to Answering Pharmacology Questions
CPR-BLS (Basic Life Support)
Electrical A&P of the Heart
54 Common Medication Prefixes and Suffixes
Advanced Cardiovascular Life Support (ACLS)
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Vitals (VS) and Assessment
Fluid Shifts (Ascites) (Pleural Effusion)
Pediatric Advanced Life Support (PALS)
Therapeutic Drug Levels (Digoxin, Lithium, Theophylline, Phenytoin)
Essential NCLEX Meds by Class
Isotonic Solutions (IV solutions)
Neonatal Resuscitation Program (NRP)
6 Rights of Medication Administration
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
The SOCK Method – Overview
The SOCK Method – S
The SOCK Method – O
The SOCK Method – C
The SOCK Method – K
Basics of Calculations
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Dimensional Analysis Nursing (Dosage Calculations/Med Math)
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Oral Medications
Chloride-Cl (Hyperchloremia, Hypochloremia)
Injectable Medications
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
IV Infusions (Solutions)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Complex Calculations (Dosage Calculations/Med Math)
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Nursing Care and Pathophysiology for Heart Failure (CHF)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Atrial Flutter
Pacemakers
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Benzodiazepines
Nursing Care and Pathophysiology of Hypertension (HTN)
Cardiac (Heart) Disease in Pregnancy
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Dehydration
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Fetal Circulation
MAOIs
SSRIs
TCAs
Congenital Heart Defects (CHD)
Defects of Increased Pulmonary Blood Flow
Defects of Decreased Pulmonary Blood Flow
Insulin
Obstructive Heart (Cardiac) Defects
Mixed (Cardiac) Heart Defects
Histamine 1 Receptor Blockers
Histamine 2 Receptor Blockers
Renin Angiotensin Aldosterone System
ACE (angiotensin-converting enzyme) Inhibitors
Angiotensin Receptor Blockers
Calcium Channel Blockers
Cardiac Glycosides
Metronidazole (Flagyl) Nursing Considerations
Ciprofloxacin (Cipro) Nursing Considerations
Vancomycin (Vancocin) Nursing Considerations
Anti-Infective – Penicillins and Cephalosporins
Atypical Antipsychotics
Autonomic Nervous System (ANS)
Sympathomimetics (Alpha (Clonodine) & Beta (Albuterol) Agonists)
Parasympathomimetics (Cholinergics) Nursing Considerations
Parasympatholytics (Anticholinergics) Nursing Considerations
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Epoetin Alfa
HMG-CoA Reductase Inhibitors (Statins)
Magnesium Sulfate
NSAIDs
Corticosteroids
Hydralazine (Apresoline) Nursing Considerations
Nitro Compounds
Vasopressin
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Base Excess & Deficit
Blood Flow Through The Heart
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Coronary Circulation
Fluid Compartments
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
Lactic Acid
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Pacemakers
Performing Cardiac (Heart) Monitoring
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Proton Pump Inhibitors
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Shock Module Intro
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)