3rd Degree AV Heart Block (Complete Heart Block)

You're watching a preview. 300,000+ students are watching the full lesson.
Maria Stewart
BSN,RN,CCRN, CMSRN
Master
To Master a topic you must score > 80% on the lesson quiz.
Take Quiz

Included In This Lesson

Study Tools For 3rd Degree AV Heart Block (Complete Heart Block)

3rd Degree (Complete) AV Block (Image)
Parts of EKG waveform (Image)
10 Common EKG Heart Rhythms (Cheatsheet)
EKG Chart (Cheatsheet)
EKG Electrical Activity Worksheet (Cheatsheet)
Heart Rhythms Signs and Symptoms (Cheatsheet)
Heart Rhythm Identification (Cheatsheet)
Heart Blocks (Cheatsheet)
NURSING.com students have a 99.25% NCLEX pass rate.

Outline

Overview

  1. 3rd degree AV heart block
    1. Complete heart block
    2. Atria are contracting at own pace
      1. Signal unable to get to the ventricles
    3. Ventricles are contracting at own slow pace
      1. Decreased CO and perfusion
    4. Dissociation between P waves and QRS complex
      1. NO relationship between the atria and ventricles

Nursing Points

General

  1. Characteristics of 3rd degree AV heart block
    1.  Rhythm
      1. Regular
        1. P to P
      2. Regular
        1. R to R
    2. Rate
      1. Varies
        1. Usually slow
          1. Ventricular rate
        2. More P waves
          1. Normal atrial rate
    3. P:QRS ratio
      1. No relationship between P waves and QRS
        1. Not measurable
    4. PR interval
      1. No relationship between P waves and QRS
        1. Not measurable
    5. QRS complex
      1. Wide
        1. >0.12 seconds

Assessment

  1. Patient presentation
    1. Fatigue
    2. Dizzy/Syncope
    3. Decreased CO
      1. Hypotensive
      2. Chest pain
  2. Medical emergency

Therapeutic Management

  1. Nursing interventions
    1. Assess patient
  2. Therapeutic management
    1. Pacemaker
      1. Temporary if unstable/emergent

Nursing Concepts

  1. EKG  rhythms
  2. Perfusion

Patient Education

  1. Seek medical help

Unlock the Complete Study System

Used by 300,000+ nursing students. 99.25% NCLEX pass rate.

200% NCLEX Pass Guarantee.
No Contract. Cancel Anytime.

Transcript

Hey guys, so we made it to our last lesson in this EKG section, we are going to finish strong and talk about another deadly arrhythmia. So we are going to talk about 3rd degree AV heart block, it is also called complete heart block. This is a deadly arrhythmia and when you see this you need to do something about it right away before our patients die. So let’s break down the characteristics of this rhythm and talk about it.

So in 3rd degree AV heart blocks the electrical conduction is unable to reach the ventricles, the SA node still initiates the impulse across the atria at a rate of 60-100 beats per minute. So what is supposed to happen is the SA node sends the impulse down to the AV node, down the Bundle of His, right and left bundle branches and purkinje fibers. In complete heart block, the signal is not getting down to the ventricles so the ventricles would not contract and the patient would die, but because the heart is so smart, it picks up its own ventricular rate. If you remember one of the first lessons where we talk about conduction, I mentioned that the SA node initiates the impulse at a rate of 60-100, if it quits working the AV node initiates the impulse at a rate of 40-60 beats per minute, and if the AV node quits working the ventricles pick up the pace at a rate of 20-40 beats per minute. That is what is happening here, since the ventricles are not receiving the signal from the atria it starts beating at its own pace with its own electrical conduction at a rate of 20-40 beats per minute. So the atria are contracting like they should and the signal does not reach the ventricles, they contract at their own pace. So there is no connection or relationship between the atria and ventricles. Since the atria and ventricles contract when they want, cardiac output is significantly decreased, something needs to be done as soon as possible. Now let’s break down each of the steps and talk about the characteristics of 3rd degree heart block.

So in step one we need to see if our rhythm is regular or irregular, and with 3rd degree heart block we need to look at it a little differently. We have more P waves than we do QRS so usually from one P wave to the other it is regular, like look at this P wave and this one and this one we have about the same number of boxes in between and if we march it out, they are regular. The ventricular rhythm can be regular but can be irregular. So the atrial rate is regular, and the ventricular rate is regular or irregular. In step 2 we need to look at the heart rate and we have 40 beats per minute, it is usually slow in 3rd degree heart block. In step 3 we look at the P:QRS ratio, there is not association between the P and QRS, and there are more P waves than QRS, the P:QRS ratio, it is not a 1 to 1 conduction. In step 4 we look at the PR interval and that is not measurable since we have more P waves than QRS. In step 5 we look at the QRS complex so from here to here we have about 3 boxes so it’s 0.12 seconds, it is usually wider in complete heart block. In step 6 would be to identify the rhythm and we have 3rd degree heart AV heart block or complete heart block. So let’s recap the characteristics, ventricular rhythm is regular or irregular, the P to P is regular. The heart rate varies but it is usually low, the P:QRS ratio is not 1 to 1 and the PR interval is not measurable and the QRS complex is wider than normal. In this rhythm you need to remember that there is a complete block from the atria to the ventricles, they are doing their own thing and there is no communication between the upper and lower chambers. So let’s talk about signs and symptoms and treatment measures.

So because the cardiac output is severely decreased in this rhythm, there clinical presentations are fatigue, dizziness, syncope, hypotension, chest pain, and they will be hemodynamically unstable. The priority nursing interventions are to assess the patient determine severity and prepare them for a pacemaker. We will do a temporary pacemaker if unstable until they get a permanent pacemaker inserted.

So the key points to take away from this lesson are to remember the abnormalities of 3rd degree AV heart block, also called complete heart block. The electrical signal from the atria is not reaching the ventricles, there is a dissociation between the atria and ventricles. The atria are contracting at their own pace and the ventricles contract at their own pace, there is no relationship between the P waves and QRS, this causes decreased cardiac output. The pain nursing interventions are to quickly identify the rhythm and prepare for a temporary pacemaker if unstable or a permanent pacemaker for a longer management.
So I hope that you guys have enjoyed this lesson and feel more comfortable identifying 3rd degree AV heart blocks or complete heart blocks. Make sure you check out all of the resources attached to this lesson. Make sure you go back and look at the last lessons regarding the heart blocks and pick out the differences between the 3 blocks so you can identify them. Now, go out and be your best self today! And, as always, happy nursing!

Study Faster with Full Video Transcripts

99.25% NCLEX Pass Rate vs 88.8% National Average

200% NCLEX Pass Guarantee.
No Contract. Cancel Anytime.

🎉 Special Back to School Sale 🎉

Ready to Stop Struggling?

NURSING.com Academy IS The Visual Learning Platform That Actually Makes Nursing Click

Sale Ends Jan 31st

NP 4 Exam 2

Concepts Covered:

  • Circulatory System
  • Urinary System
  • Adult
  • Basic
  • Test Taking Strategies
  • Prefixes
  • Suffixes
  • Integumentary Disorders
  • Respiratory Disorders
  • Pediatric
  • Bipolar Disorders
  • Immunological Disorders
  • Labor Complications
  • Neonatal
  • Medication Administration
  • Disorders of Pancreas
  • Pregnancy Risks
  • Cardiac Disorders
  • Learning Pharmacology
  • Eating Disorders
  • Dosage Calculations
  • Emergency Care of the Cardiac Patient
  • Substance Abuse Disorders
  • Vascular Disorders
  • Endocrine and Metabolic Disorders
  • Shock
  • Fetal Development
  • Depressive Disorders
  • Anxiety Disorders
  • Cardiovascular Disorders
  • Liver & Gallbladder Disorders
  • Upper GI Disorders
  • Female Reproductive Disorders
  • Neurologic and Cognitive Disorders
  • Personality Disorders
  • Nervous System
  • Urinary Disorders
  • Hematologic Disorders
  • Disorders of the Posterior Pituitary Gland
  • Respiratory System
  • Renal Disorders
  • Noninfectious Respiratory Disorder
  • Shock

Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Life Support Review Course Introduction
12 Points to Answering Pharmacology Questions
CPR-BLS (Basic Life Support)
Electrical A&P of the Heart
54 Common Medication Prefixes and Suffixes
Advanced Cardiovascular Life Support (ACLS)
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Vitals (VS) and Assessment
Fluid Shifts (Ascites) (Pleural Effusion)
Pediatric Advanced Life Support (PALS)
Therapeutic Drug Levels (Digoxin, Lithium, Theophylline, Phenytoin)
Essential NCLEX Meds by Class
Isotonic Solutions (IV solutions)
Neonatal Resuscitation Program (NRP)
6 Rights of Medication Administration
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
The SOCK Method – Overview
The SOCK Method – S
The SOCK Method – O
The SOCK Method – C
The SOCK Method – K
Basics of Calculations
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Dimensional Analysis Nursing (Dosage Calculations/Med Math)
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Oral Medications
Chloride-Cl (Hyperchloremia, Hypochloremia)
Injectable Medications
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
IV Infusions (Solutions)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Complex Calculations (Dosage Calculations/Med Math)
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Nursing Care and Pathophysiology for Heart Failure (CHF)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Atrial Flutter
Pacemakers
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Benzodiazepines
Nursing Care and Pathophysiology of Hypertension (HTN)
Cardiac (Heart) Disease in Pregnancy
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Dehydration
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Fetal Circulation
MAOIs
SSRIs
TCAs
Congenital Heart Defects (CHD)
Defects of Increased Pulmonary Blood Flow
Defects of Decreased Pulmonary Blood Flow
Insulin
Obstructive Heart (Cardiac) Defects
Mixed (Cardiac) Heart Defects
Histamine 1 Receptor Blockers
Histamine 2 Receptor Blockers
Renin Angiotensin Aldosterone System
ACE (angiotensin-converting enzyme) Inhibitors
Angiotensin Receptor Blockers
Calcium Channel Blockers
Cardiac Glycosides
Metronidazole (Flagyl) Nursing Considerations
Ciprofloxacin (Cipro) Nursing Considerations
Vancomycin (Vancocin) Nursing Considerations
Anti-Infective – Penicillins and Cephalosporins
Atypical Antipsychotics
Autonomic Nervous System (ANS)
Sympathomimetics (Alpha (Clonodine) & Beta (Albuterol) Agonists)
Parasympathomimetics (Cholinergics) Nursing Considerations
Parasympatholytics (Anticholinergics) Nursing Considerations
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Epoetin Alfa
HMG-CoA Reductase Inhibitors (Statins)
Magnesium Sulfate
NSAIDs
Corticosteroids
Hydralazine (Apresoline) Nursing Considerations
Nitro Compounds
Vasopressin
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Base Excess & Deficit
Blood Flow Through The Heart
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Coronary Circulation
Fluid Compartments
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
Lactic Acid
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Pacemakers
Performing Cardiac (Heart) Monitoring
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Proton Pump Inhibitors
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Shock Module Intro
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)