Respiratory Acidosis (interpretation and nursing interventions)

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Nichole Weaver
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Included In This Lesson

Study Tools For Respiratory Acidosis (interpretation and nursing interventions)

63 Must Know Lab Values (Book)
Respiratory Acidosis Assessment (Picmonic)
Respiratory Acidosis Interventions (Picmonic)
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Outline

Overview

    1. Lab Values
      1. LOW pH (< 7.35)
      2. HIGH PaCO2 (> 45 mmHg)
      3. May also see low oxygenation
        1. LOW PaO2
        2. LOW SpO2

Nursing Points

General

  1. Causes
    1. Hypoventilation – slow or shallow respirations
      1. Sedative or narcotic overdose
      2. Brain injury
    2. Airway obstructions
      1. Asthma
      2. COPD
      3. Aspiration
    3. Lung collapse
      1. Atelectasis
      2. Pneumo/Hemothorax

Assessment

  1. Symptoms
    1. Signs of the cause
    2. Decreased LOC
      1. Confusion
      2. Restlessness
    3. Dyspnea
    4. Headache
    5. Muscle weakness
    6. Arrhythmias

Respiratory Acidosis Nursing Interventions

  1. Address the cause
  2. Provide supplemental oxygen
  3. Airway Support
    1. Bronchodilators
    2. Artificial airways
  4. Assisted Ventilation
    1. Noninvasive ventilation
      1. CPAP
      2. BiPAP
    2. Invasive ventilation
      1. Intubation (ETT)
  5. Reversal of Drug Overdose
    1. Naloxone
    2. Flumazenil

Nursing Concepts

  1. Acid-Base Balance
  2. Gas Exchange
  3. Oxygenation

Patient Education

  1. Proper dosing for narcotics, sedatives → signs of overdose to watch for
  2. Use of inhalers in asthma/COPD
  3. Purpose for mechanical ventilation

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Transcript

Now that we have reviewed how to interpret the acid-base portion of an arterial blood gas, we are going to start diving into the four specific conditions and what causes them and how they present. The first one we will look at is respiratory acidosis.

The lab values that you’ll see in respiratory acidosis are a low pH and a high PaCO2. Because of the conditions associated with respiratory acidosis, it is very likely that you will also see some hypoxemia evidenced by a low PaO2 and a low SaO2

So, the number one thing that causes respiratory acidosis is retention of carbon dioxide. Remember that we breathe out carbon dioxide with every breath. Think of it like the pressure release valve on water heater. When the pressure gets too high the release valve opens to let out some of the steam. Our lungs do the same thing when carbon dioxide gets too high. If anything causes that pressure release valve to be blocked or to not work appropriately, that carbon dioxide will get stuck inside body. The most common cause is hypoventilation. Hypoventilation could be a low respiratory rate or very shallow respirations or both. Less breathing means less CO2 being blown off. This happens quite frequently with any kind of sedative or opioid overdose. Other possible causes of CO2 retention are anything that obstructs airflow or prevents expansion of the lungs. Remember, it’s all about gas exchange, so anything that prevents proper gas exchange could cause us to retain carbon dioxide. This could be aspiration, asthma, COPD, or bronchospasm obstructing flow. Or, it could be some form of lung collapse like atelectasis, or a hemo or pneumothorax preventing expansion of the lung. So any of these things can cause CO2 to be built up in our system. And remember that CO2 is an acid. so more CO2 means more acid, hence respiratory acidosis.

The number one thing that you’re going to assess in a patient with respiratory acidosis is signs and symptoms of the cause of the acidosis. So you may see decreased lung sounds if they have a pneumothorax, or decreased level of consciousness if they have overdosed on a sedative or an opioid. Make sure that you are thoroughly assessing those things in addition to the signs of the acidosis itself. Those signs would be things like decreased level of consciousness, confusion, and commonly they get quite restless or anxious. Remember that the brain is VERY greedy and VERY sensitive to changes in pH and CO2 levels. They may also have trouble breathing or have a headache. And, it is also possible with acidosis that we will see vomiting. It’s a way for the body to get rid of excess circulating acids. However, because this is a respiratory source, it is much less common than it is with a metabolic acidosis. The other things we will see are muscle weakness and arrhythmias related to the hyperkalemia. Remember that the excess hydrogen ions in acidosis will attempt to switch places with the potassium inside the cell. That kicks extra potassium out into the bloodstream causing hyperkalemia. So, you will see signs of the cause, signs of the acidosis, and possibly signs of hyperkalemia.

  • What are the nursing interventions for respiratory acidosis?
    • Address the cause, Provide supplemental oxygen, Airway Support via bronchodilators, artificial airways, and ventilation.

Management of pretty much every acid-base imbalance will always start with fixing the cause. We need to protect and open up their airways if they have some kind of obstruction. This might mean bronchodilators or an artificial airway. We also may need to provide assistance with ventilation. This could be non-invasive ventilation like BiPAP or CPAP or it could be invasive ventilation where we intubate the patient and place them on a mechanical ventilator. Either way those things are going to help facilitate the gas exchange and control the CO2 excretion a bit better. If they have signs of a possible drug overdose, then we need to reverse that drug overdose. Opioids get reversed with naloxone, and benzodiazepines get reversed with flumazenil. Make sure you know what your patient has going on specifically and do a detailed assessment so you know how to proceed to fix the problem.

Priority nursing concepts for a patient with respiratory acidosis will be, of course, acid-base balance. Also gas exchange and oxygenation. Remember that the whole process of exchanging carbon dioxide for oxygen occurs in the lungs. if we aren’t getting the CO2 out, we probably aren’t getting much oxygen in either.

So let’s recap. The lab values that you’ll see in a respiratory acidosis are a low ph and a high PaCO2, plus also possible low oxygenation values as well. Common causes are things that will create retention of carbon dioxide like hypoventilation, airway obstruction, and lung collapse. When you assess the patient you will see signs of the cause, signs of acidosis like decreased LOC, and possibly vomiting, and signs of hyperkalemia like arrhythmias. To manage respiratory acidosis, our number one priority is to support their ventilation and oxygenation, and to identify and treat the cause.

Remember that providing supplemental oxygen to someone who is not breathing correctly or has an airway obstruction is not beneficial. Make sure that their Airway is open and that their breathing is appropriate, and then provide oxygen. Yes, in the real world it only takes 5 Seconds to apply oxygen. However, it also only takes 5 Seconds to apply an EKG lead, but that is not going to help the patient. It’s not always about how quickly you can do something, but about the impact it’s actually going to have. So make sure that your patients airway and breathing are taking care of first.

So, that’s it for respiratory acidosis. I’ve attached the ARDS case study, because it’s a classic example of respiratory acidosis. So make sure you check that out, as well as all of the other resources attached to this lesson, and that you check out the next 3 lessons to learn about the other acid-base imbalances too! Now, go out and be your best selves today. And, as always, happy nursing!!

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NP 4 Exam 2

Concepts Covered:

  • Circulatory System
  • Urinary System
  • Adult
  • Basic
  • Test Taking Strategies
  • Prefixes
  • Suffixes
  • Integumentary Disorders
  • Respiratory Disorders
  • Pediatric
  • Bipolar Disorders
  • Immunological Disorders
  • Labor Complications
  • Neonatal
  • Medication Administration
  • Disorders of Pancreas
  • Pregnancy Risks
  • Cardiac Disorders
  • Learning Pharmacology
  • Eating Disorders
  • Dosage Calculations
  • Emergency Care of the Cardiac Patient
  • Substance Abuse Disorders
  • Vascular Disorders
  • Endocrine and Metabolic Disorders
  • Shock
  • Fetal Development
  • Depressive Disorders
  • Anxiety Disorders
  • Cardiovascular Disorders
  • Liver & Gallbladder Disorders
  • Upper GI Disorders
  • Female Reproductive Disorders
  • Neurologic and Cognitive Disorders
  • Personality Disorders
  • Nervous System
  • Urinary Disorders
  • Hematologic Disorders
  • Disorders of the Posterior Pituitary Gland
  • Respiratory System
  • Renal Disorders
  • Noninfectious Respiratory Disorder
  • Shock

Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Life Support Review Course Introduction
12 Points to Answering Pharmacology Questions
CPR-BLS (Basic Life Support)
Electrical A&P of the Heart
54 Common Medication Prefixes and Suffixes
Advanced Cardiovascular Life Support (ACLS)
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Vitals (VS) and Assessment
Fluid Shifts (Ascites) (Pleural Effusion)
Pediatric Advanced Life Support (PALS)
Therapeutic Drug Levels (Digoxin, Lithium, Theophylline, Phenytoin)
Essential NCLEX Meds by Class
Isotonic Solutions (IV solutions)
Neonatal Resuscitation Program (NRP)
6 Rights of Medication Administration
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
The SOCK Method – Overview
The SOCK Method – S
The SOCK Method – O
The SOCK Method – C
The SOCK Method – K
Basics of Calculations
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Dimensional Analysis Nursing (Dosage Calculations/Med Math)
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Oral Medications
Chloride-Cl (Hyperchloremia, Hypochloremia)
Injectable Medications
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
IV Infusions (Solutions)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Complex Calculations (Dosage Calculations/Med Math)
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Nursing Care and Pathophysiology for Heart Failure (CHF)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Atrial Flutter
Pacemakers
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Benzodiazepines
Nursing Care and Pathophysiology of Hypertension (HTN)
Cardiac (Heart) Disease in Pregnancy
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Dehydration
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Fetal Circulation
MAOIs
SSRIs
TCAs
Congenital Heart Defects (CHD)
Defects of Increased Pulmonary Blood Flow
Defects of Decreased Pulmonary Blood Flow
Insulin
Obstructive Heart (Cardiac) Defects
Mixed (Cardiac) Heart Defects
Histamine 1 Receptor Blockers
Histamine 2 Receptor Blockers
Renin Angiotensin Aldosterone System
ACE (angiotensin-converting enzyme) Inhibitors
Angiotensin Receptor Blockers
Calcium Channel Blockers
Cardiac Glycosides
Metronidazole (Flagyl) Nursing Considerations
Ciprofloxacin (Cipro) Nursing Considerations
Vancomycin (Vancocin) Nursing Considerations
Anti-Infective – Penicillins and Cephalosporins
Atypical Antipsychotics
Autonomic Nervous System (ANS)
Sympathomimetics (Alpha (Clonodine) & Beta (Albuterol) Agonists)
Parasympathomimetics (Cholinergics) Nursing Considerations
Parasympatholytics (Anticholinergics) Nursing Considerations
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Epoetin Alfa
HMG-CoA Reductase Inhibitors (Statins)
Magnesium Sulfate
NSAIDs
Corticosteroids
Hydralazine (Apresoline) Nursing Considerations
Nitro Compounds
Vasopressin
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Base Excess & Deficit
Blood Flow Through The Heart
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Coronary Circulation
Fluid Compartments
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
Lactic Acid
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Pacemakers
Performing Cardiac (Heart) Monitoring
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Proton Pump Inhibitors
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Shock Module Intro
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)