Nursing Care and Pathophysiology for Asthma

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Brad Bass
ASN,RN
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Included In This Lesson

Study Tools For Nursing Care and Pathophysiology for Asthma

Asthma management (Mnemonic)
Asthma Pathochart (Cheatsheet)
Asthma Medications (Cheatsheet)
Asthma Diagram (Image)
Asthma Assessment (Picmonic)
Asthma Implementation and Education (Picmonic)
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Outline

Pathophysiology: Asthma is a respiratory condition with chronic inflammation of the bronchioles and bronchoconstriction. This causes airway restriction. Asthma attacks are caused by triggers such as infections, allergens, exercise, and other irritants.

Overview

  1. Inflammatory disorder of airways
  2. Stimulated by triggers (infection, allergens, exercise, irritants)
  3. Status Asthmaticus – life-threatening condition
    1. Asthma unresponsive to treatment

Nursing Points

General

  1. Narrowed airways = ↓ gas exchange
    1. Inflammation of airways
    2. Bronchoconstriction
    3. Excessive mucus production

Assessment

  1. Symptoms
    1. Wheezing/crackles
    2. Restless/anxious
    3. Diminished breath sounds
    4. tachypnea
  2. Diagnostics
    1. Peak Flow Rate
      1. Volume of expired air
      2. Patient should track and know baseline
      3. Stable = 80-100% baseline
      4. Caution = 50-80% baseline
      5. Danger = <50% baseline
    2. Pulmonary Function Tests
    3. X-ray to rule out other causes

Therapeutic Management

  1. High-fowler’s or position of comfort
  2. Administer O2
  3. Medications
    1. Epi-Pen if allergic reaction
    2. Bronchodilators
    3. Corticosteroids
    4. Leukotriene Modulators

Nursing Concepts

  1. Oxygenation
    1. Listen to lungs
    2. Monitor SpO2
    3. Administer supplemental O2
  2. Gas Exchange
    1. Monitor ABG
    2. Monitor for s/s CO2 toxicity
      1. ↓ LOC
  3. Comfort
    1. Keep patient calm
    2. Encourage position of comfort

Patient Education

  1. Identify Triggers and Avoid
    1. Allergy tests
    2. Smoke / Secondhand smoke
      1. SMOKING CESSATION
    3. Keep a journal
  2. Proper use of Inhaler
    1. Shake 10-15 times
    2. Large breathe, exhale completely
    3. Mouthpiece in mouth, seal with lips
    4. Tilt head back to open airway
    5. Depress inhaler, slow, deep breath in
    6. Hold breath 5-10 sec
    7. Breathe out slow
    8. Repeat if 2nd puff ordered
    9. Use spacer if needed
  3. Peak Flow Test
    1. Daily testing – perform 3 times and record best effort
    2. Track in a journal
    3. Report to provider if in caution or danger zone

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Transcript

Hey guys, my name is Brad and welcome to nursing.com. And in today’s video, what we’re going to be discussing is asthma. We’re going to dive into some of the patho behind asthma, as well as some signs and symptoms that we may see in our patients, as well as how we’re going to treat. Without further ado, let’s dive in.

Regarding the patho of asthma and patients who have asthma, what they have are super, sensitive airways, right? Very, very, sensitive airways. Asthma is a genetically acquired condition. And in patients who have asthma, they have hypersensitivity of these airways, right? These bronchi, these bronchioles that come down and feed into these alveoli are very, very, sensitive. So what occurs in asthma is a patient breathes in some sort of irritants, right? Dust debris, we’ll get into some of the possible irritants, but they breathe in something. Let’s call it dust for this instance. Dust gets breathed into the lungs and because these lungs are so sensitive, they pick up on it immediately, and a cascade of events actually ends up occurring as a result, right? The first thing that happens is what we have is actual contraction of this bronchial smooth muscle, right? We actually have contraction of the smooth muscle, which causes narrowing of that inner lumen, right, causes tightening of the airways, constriction of the airways. The second thing that occurs, because these patient’s lungs, that are hypersensitive, view that dust or debris that they just inhale as a foreign attack on the lungs. They’re that sensitive. This is now a foreign attack on the lungs. What actually ends up occurring is we have an inflammatory response, an inflammatory cascade that rushes to the lungs to try and attack whatever this foreign body is, this dirt, this debris. And so we’re going to actually see inflammation of the airways in addition to that constriction. And the other thing that I left here is, actual hypersecretion of mucus within the airways as well, right? We’ve breathed, we’ve inhaled dust. We want to try and trap that dust, so we secrete extra mucus to try and trap that dust, our smooth muscle contracts and constricts those airways. And then we have an insult to our lungs. So we release an inflammatory cascade causing inflammation of our airways, right? Kind of a three-pronged reaction to the inhalation of some sort of irritant in these hypersensitive airways. So the importance of this slide is just for you guys, to be able to familiarize yourself with some of these triggers for asthma, right? There are a ton of different kinds. Smoke is a really, really big one, right? Heavy fragrance, fragrances, such as perfume. Pollen, right? This is a really, really big one that can trigger an actual asthma attack. Pet dander, dust, actually, cold air, also stress, can cause asthma attacks. The entire idea is that all of these end up triggering that very, very sensitive airway to produce that three-pronged attack, that three-pronged cascade of symptoms of pathophys that we just discussed.

Regarding some assessment findings in patients with asthma, remember, right, we have this incredibly narrow airway that is constricted, that is inflamed, because we’ve inhaled some sort of foreign body. And that is just absolutely dripping with mucus. So patients are going to have an incredibly difficult time getting air in, getting oxygen in. Therefore, they’re going to be short of breath. Therefore, they’re going to have low O2 Sats. Therefore, they’re going to be tachycardic, right? That heart is going to try and compensate by pumping oxygen containing blood up to the lungs. We’re trying to get 02, we’re starving for O2. Because we’re starving for 02 and we’re so short of breath, patients can become anxious. Patients can become confused, right? We may end up seeing decreased levels of consciousness as we have alterations in our blood pH. Make sure you check out our ABGs video if you’re a little bit unfamiliar on respiratory acidosis, respiratory alkalosis, it may make a little bit more sense. Also, adventitious lung sounds. Speaking of making sense, this makes sense that what we’re going to hear is wheezing. As we’re trying to pass air through an incredibly narrow pipe, that we’re going to hear crackles, as we’re trying to pass air through all of this thick mucus, peak expiratory flow, as well as chest x-rays that we’re going to be getting on our patients to keep an eye on the progression of their asthma. 

How are we going to treat our patients with asthma? Well, of course, if our patient has an asthma attack, if they have that hypersensitive reaction due to exposure to some allergen, of course remove the patient from the allergen. We want to make sure that we are giving patients rescue inhalers should they need them for instances where they have these acute flare ups of asthma attacks, right? Things such as albuterol, that’s going to be the most common rescue inhaler you’re going to be familiar with, right? The entire idea is these airways are so constricted, we need to dilate them. So let’s bronchodilate them with rescue inhalers, such as albuterol. And speaking of bronchodilators, that’s the next one, right? Things such as beta-2 agonists, right? Things that actually stimulate the beta-2 cells in the lungs themselves, in order to produce this bronchodilation.  Things such as formoterol, if I can spell that. Steroids, right? Remember that three-pronged attack that our lungs are being hit with in asthma. Not only are we having this bronchoconstriction, but we’re also having the inflammation from this inflammatory response from this pathogen that we’ve inhaled. So we want to give steroids to decrease this inflammation. Also, 02, again, that’s a no brainer. Patients starving for 02, we need to give them supplemental oxygen. And also making sure that we sit our patient up. You may also have heard it being called something like a tripod position. Basically sit that patient upright in bed to allow those lungs to fully expand, to allow them to get as much oxygen in as they can. 

Now, some education that we’re going to give our patient, how to properly use these inhalers, these rescue inhalers albuterol, making sure that they’re very familiar with how to use them, so that should an asthma attack occur, they’re more calm and prepared on how to use them. Medication adherence, those bronchodilators, those steroids, things that we’re going to use to try and treat that three-prong attack, right? Also, avoiding allergens, avoiding irritants, making sure that they’re familiar with what triggers their asthma. Peak expiratory flow recordings. Remember we mentioned peak expiratory flow. Basically, this is something that a patient’s going to do, periodically, to measure the amount of expiratory flow that they can actually exhale. And this is just the way in which patients are able to measure the severity of their asthma. And also educating them on signs and symptoms of asthma attacks as well as when to seek help. 

So summarizing some key points from asthma. Remember in patients who have asthma, they have super sensitive airways and it’s this hypersensitivity through the inhalation of some sort of foreign body, some sort of dust or debris that ends up leading to this three-pronged attack that causes so many problems for patients with asthma. Remembering and familiarizing yourself with those different triggers that cause the asthma attacks, knowing those various assessments are all related to that bronchoconstriction, that inflammatory response, as well as that hypersecretion of mucus inside of those airways, the therapeutic management focused on combating those three-pronged attack, those three things that are causing such issues in patients with asthma, as well as the education, which we just discussed. 

Guys, that was asthma. And I really hope that this information helps you as you move forward through nursing school, helps you as you move forward taking those exams, and helps you as you take care of those patients. Go out there and be your best selves today. And as always, happy nursing.

 

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NP4 exam1

Concepts Covered:

  • Circulatory System
  • Urinary System
  • Noninfectious Respiratory Disorder
  • Respiratory System
  • Integumentary Disorders
  • Respiratory Disorders
  • Labor Complications
  • Disorders of Pancreas
  • Pregnancy Risks
  • Cardiac Disorders
  • Eating Disorders
  • Respiratory Emergencies
  • Infectious Respiratory Disorder
  • Emergency Care of the Cardiac Patient
  • Vascular Disorders
  • Shock
  • Medication Administration
  • Upper GI Disorders
  • Fundamentals of Emergency Nursing
  • Understanding Society
  • Adulthood Growth and Development
  • Oncologic Disorders
  • Postoperative Nursing
  • Renal Disorders
  • Microbiology
  • Intraoperative Nursing
  • Shock
  • Tissues and Glands
  • Newborn Care

Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Respiratory Course Introduction
Electrical A&P of the Heart
Respiratory A&P Module Intro
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Lung Sounds
Alveoli & Atelectasis
Alveoli & Atelectasis
Fluid Shifts (Ascites) (Pleural Effusion)
Gas Exchange
Gas Exchange
Isotonic Solutions (IV solutions)
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
Lung Diseases Module Intro
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology for Asthma
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of COPD (Chronic Obstructive Pulmonary Disease)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of COPD (Chronic Obstructive Pulmonary Disease)
Chloride-Cl (Hyperchloremia, Hypochloremia)
Restrictive Lung Diseases (Pulmonary Fibrosis, Neuromuscular Disorders)
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Nursing Care and Pathophysiology of Acute Respiratory Distress Syndrome (ARDS)
Nursing Care and Pathophysiology for Pulmonary Edema
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Respiratory Infections Module Intro
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Influenza (Flu)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Nursing Care and Pathophysiology for Tuberculosis (TB)
Atrial Flutter
Pacemakers
Nursing Care and Pathophysiology of Pneumonia
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Coronavirus (COVID-19) Nursing Care and General Information
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Oxygen Delivery Module Intro
Hierarchy of O2 Delivery
Nursing Care and Pathophysiology of Hypertension (HTN)
Artificial Airways
Artificial Airways
Airway Suctioning
Airway Suctioning
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Respiratory Trauma Module Intro
Blunt Chest Trauma
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Chest Tube Management
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Pulmonary Embolism
Respiratory Procedures Module Intro
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Bariatric: IV Insertion
Base Excess & Deficit
Blood Flow Through The Heart
Bronchoscopy
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Chest Tube Management
Combative: IV Insertion
Coronary Circulation
Dark Skin: IV Insertion
Drawing Blood from the IV
Fluid Compartments
Geriatric: IV Insertion
Giving Medication Through An IV Set Port
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
How to Remove (discontinue) an IV
How to Secure an IV (chevron, transparent dressing)
Isolation Precautions (MRSA, C. Difficile, Meningitis, Pertussis, Tuberculosis, Neutropenia)
IV Catheter Selection (gauge, color)
IV Complications (infiltration, phlebitis, hematoma, extravasation, air embolism)
IV Drip Administration & Safety Checks
IV Drip Therapy – Medications Used for Drips
IV Insertion Angle
IV Insertion Course Introduction
IV Placement Start To Finish (How to Start an IV)
Lactic Acid
Lung Sounds
Maintenance of the IV
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Needle Safety
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Asthma
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Influenza (Flu)
Nursing Care and Pathophysiology for Pneumothorax & Hemothorax
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Tuberculosis (TB)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Nursing Care and Pathophysiology of Pneumonia
Pacemakers
Performing Cardiac (Heart) Monitoring
Positioning
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Selecting THE vein
Shock Module Intro
Supplies Needed
Tattoos IV Insertion
Thoracentesis
Tips & Tricks
Tips & Advice for Newborns (Neonatal IV Insertion)
Tips & Advice for Pediatric IV
Understanding All The IV Set Ports
Using Aseptic Technique
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)
Vent Alarms