Coronary Circulation

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Jon Haws
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Included In This Lesson

Study Tools For Coronary Circulation

Coronary Arteries – Location (Mnemonic)
Coronary Circulation (Cheatsheet)
Coronary Anatomy (Image)
Coronary Arteries (Picmonic)
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Outline

Overview

Heart has its own system of arteries to supply blood to myocardium

Nursing Points

General

  1. Coronary circulation branches off aorta above aortic valve
  2. Coronary perfusion during diastole
  3. Aorta
    1. Right Coronary Artery (RCA)
      1. Right Posterior Descending
      2. Right Marginal Artery
      3. SUPPLIES:
        1. RA
        2. RV
        3. Septum
        4. SA node
        5. AV node
    2. Left Main Coronary Artery (LMCA)
      1. Left Anterior Descending (LAD)
      2. Left Circumflex (LCX)
      3. SUPPLIES:
        1. LA
        2. LV
        3. Septum

Assessment

  1. Tachycardia
    1. Decreased diastolic time
    2. Decreased coronary perfusion
    3. High risk for ischemia / infarction

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Transcript

This lesson will be a quick review of coronary circulation – that is the the flow of blood to the heart muscle itself.
So, just as the heart is going to supply blood to the body, it also has to supply blood to itself. The heart is an enormous muscle and it requires a tremendous amount of cardiac output in order to just carry out its normal functions. So, because of that, the heart actually has its own system of arteries.

As you can see, the coronary arteries actually branch off of the aorta right above the aortic valve. That’s important to note, we’ll look at that in a second. On both sides of the heart you have a main coronary artery branching off – the Right Coronary Artery or RCA, and the Left Main Coronary Artery or LMCA. From there they each have smaller branches to supply the front and back of the heart. On the right we have the Right Posterior Descending which wraps around the back and the Right Marginal Artery which wraps around the front. On the left, we have the Left Anterior Descending or LAD that supplies the front left side of the heart and the Left Circumflex, often written LCX, which wraps around the back left side of the heart. You’ll notice there are more branches on the left, that’s because it is larger than the right. The main ones that are susceptible to blockage are the RCA, LMCA, LAD, and LCX – you’ll learn more about what a blockage means in the Myocardial Infarction lesson.

Now, it’s important to note what each of these coronary systems supplies blood to. On the left, it supplies the Left Atrium, Left Ventricle, and part of the Septum. On the right it supplies the Right Atrium, Right Ventricle, and part of the Septum as well, but it ALSO supplies the SA and AV node. So you can imagine that if someone had a blockage on the right side of their coronary perfusion, they might be susceptible to arrhythmias.

One more thing that’s important to note about how and when the heart perfuses itself. Remember how I said that the coronary circulation branches right above the aortic valve? Well if you think about systole and diastole. The heart contracts in systole and the Aortic Valve opens – when it does, it will briefly block the coronary circulation. Then the heart relaxes in diastole, the aortic valve closes, and blood flows through the coronary arteries. There is also a pause after diastole to allow filling of the ventricles, so this gives even more time for blood to flow to the coronary arteries. So, the heart perfuses the body during systole, and perfuses itself during diastole.

Time for some critical thinking! If my patient’s heart rate is extremely fast, what happens to their time in diastole? It decreases! Instead of Lub, Dub……..Lub, Dub…..Lub, Dub….. it becomes LubDubLubDubLubDub with no pauses in between. So if my heart perfuses itself during diastole, what happens to coronary perfusion in severe or prolonged tachycardia? It decreases dramatically!
It’s like pressing a water fountain button over and over really fast and expecting to be able to get a full drink of water. It’s not possible. You have to hold it. Without that diastolic pause, the heart struggles to perfuse itself.
In the ICU when we see a patient in a significant tachycardia (for example Supraventricular Tachycardia in the 160’s), who is stable so far, we all tend to say something like “can’t stay like that forever” or “his heart will give out eventually” because we know that the patient’s coronary perfusion is decreased significantly. So that’s just something I want you to know so that if you have a patient who’s been severely tachycardic for a long time, you will know how important it is to address the situation!
So remember, the coronary circulation is how the heart gets blood flow to itself. Both sides of the heart have a main coronary artery and then branches to cover the whole muscle. The heart perfuses the body during systole, but perfuses itself during diastole. And prolonged tachycardia can lead to decreased coronary perfusion and even a myocardial infarction.

I hope you learned something today! Go out and be your best self and as always, happy nursing!

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NP4 exam1

Concepts Covered:

  • Circulatory System
  • Urinary System
  • Noninfectious Respiratory Disorder
  • Respiratory System
  • Integumentary Disorders
  • Respiratory Disorders
  • Labor Complications
  • Disorders of Pancreas
  • Pregnancy Risks
  • Cardiac Disorders
  • Eating Disorders
  • Respiratory Emergencies
  • Infectious Respiratory Disorder
  • Emergency Care of the Cardiac Patient
  • Vascular Disorders
  • Shock
  • Medication Administration
  • Upper GI Disorders
  • Fundamentals of Emergency Nursing
  • Understanding Society
  • Adulthood Growth and Development
  • Oncologic Disorders
  • Postoperative Nursing
  • Renal Disorders
  • Microbiology
  • Intraoperative Nursing
  • Shock
  • Tissues and Glands
  • Newborn Care

Study Plan Lessons

EKG (ECG) Course Introduction
Fluid & Electrolytes Course Introduction
Respiratory Course Introduction
Electrical A&P of the Heart
Respiratory A&P Module Intro
Electrolytes Involved in Cardiac (Heart) Conduction
Fluid Pressures
Lung Sounds
Alveoli & Atelectasis
Alveoli & Atelectasis
Fluid Shifts (Ascites) (Pleural Effusion)
Gas Exchange
Gas Exchange
Isotonic Solutions (IV solutions)
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Preload and Afterload
Performing Cardiac (Heart) Monitoring
Lung Diseases Module Intro
The EKG (ECG) Graph
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology for Asthma
EKG (ECG) Waveforms
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Calculating Heart Rate
Nursing Care and Pathophysiology of COPD (Chronic Obstructive Pulmonary Disease)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of COPD (Chronic Obstructive Pulmonary Disease)
Chloride-Cl (Hyperchloremia, Hypochloremia)
Restrictive Lung Diseases (Pulmonary Fibrosis, Neuromuscular Disorders)
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Nursing Care and Pathophysiology of Acute Respiratory Distress Syndrome (ARDS)
Nursing Care and Pathophysiology for Pulmonary Edema
Phosphorus-Phos
Normal Sinus Rhythm
Normal Sinus Rhythm
Respiratory Infections Module Intro
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Influenza (Flu)
Sinus Bradycardia
Sinus Bradycardia
Sinus Tachycardia
Sinus Tachycardia
Nursing Care and Pathophysiology for Tuberculosis (TB)
Atrial Flutter
Pacemakers
Nursing Care and Pathophysiology of Pneumonia
Atrial Fibrillation (A Fib)
Atrial Fibrillation (A Fib)
Coronavirus (COVID-19) Nursing Care and General Information
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Oxygen Delivery Module Intro
Hierarchy of O2 Delivery
Nursing Care and Pathophysiology of Hypertension (HTN)
Artificial Airways
Artificial Airways
Airway Suctioning
Airway Suctioning
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Respiratory Trauma Module Intro
Blunt Chest Trauma
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Chest Tube Management
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Pulmonary Embolism
Respiratory Procedures Module Intro
ABG (Arterial Blood Gas) Interpretation-The Basics
ABG (Arterial Blood Gas) Oxygenation
ABG Course (Arterial Blood Gas) Introduction
ABGs Nursing Normal Lab Values
ABGs Tic-Tac-Toe interpretation Method
Acute Coronary Syndrome (ACS) Module Intro
Bariatric: IV Insertion
Base Excess & Deficit
Blood Flow Through The Heart
Bronchoscopy
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Course Introduction
Cardiovascular Disorders (CVD) Module Intro
Chest Tube Management
Combative: IV Insertion
Coronary Circulation
Dark Skin: IV Insertion
Drawing Blood from the IV
Fluid Compartments
Geriatric: IV Insertion
Giving Medication Through An IV Set Port
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Hemodynamics
Hemodynamics
How to Remove (discontinue) an IV
How to Secure an IV (chevron, transparent dressing)
Isolation Precautions (MRSA, C. Difficile, Meningitis, Pertussis, Tuberculosis, Neutropenia)
IV Catheter Selection (gauge, color)
IV Complications (infiltration, phlebitis, hematoma, extravasation, air embolism)
IV Drip Administration & Safety Checks
IV Drip Therapy – Medications Used for Drips
IV Insertion Angle
IV Insertion Course Introduction
IV Placement Start To Finish (How to Start an IV)
Lactic Acid
Lung Sounds
Maintenance of the IV
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
MI Surgical Intervention
Needle Safety
Nursing Care and Pathophysiology for Aortic Aneurysm
Nursing Care and Pathophysiology for Arterial Disorders
Nursing Care and Pathophysiology for Asthma
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cardiomyopathy
Nursing Care and Pathophysiology for Distributive Shock
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Influenza (Flu)
Nursing Care and Pathophysiology for Pneumothorax & Hemothorax
Nursing Care and Pathophysiology for Thrombophlebitis (clot)
Nursing Care and Pathophysiology for Tuberculosis (TB)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis
Nursing Care and Pathophysiology of Pneumonia
Pacemakers
Performing Cardiac (Heart) Monitoring
Positioning
Potassium-K (Hyperkalemia, Hypokalemia)
Preload and Afterload
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
ROME – ABG (Arterial Blood Gas) Interpretation
Selecting THE vein
Shock Module Intro
Supplies Needed
Tattoos IV Insertion
Thoracentesis
Tips & Tricks
Tips & Advice for Newborns (Neonatal IV Insertion)
Tips & Advice for Pediatric IV
Understanding All The IV Set Ports
Using Aseptic Technique
Venous Disorders (Chronic venous insufficiency, Deep venous thrombosis/DVT)
Vent Alarms