Nursing Care Plan (NCP) for Pulmonary Embolism

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Study Tools For Nursing Care Plan (NCP) for Pulmonary Embolism

Thrombophlebitis – Virchow’s Triad (Cheatsheet)
Pulmonary Embolism Assessment (Picmonic)
Example Care Plan_Pulmonary Embolism (Cheatsheet)
Blank Nursing Care Plan_CS (Cheatsheet)
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Outline

Lesson Objective for Pulmonary Embolism Nursing Care Plan

 

  • Early Recognition and Intervention:
    • Educate healthcare providers on the early signs and symptoms of pulmonary embolism (PE) to facilitate prompt recognition and immediate initiation of life-saving interventions.
  • Comprehensive Patient Assessment:
    • Train nursing staff to conduct thorough and timely assessments of patients at risk for or presenting with symptoms suggestive of PE, focusing on respiratory status, cardiovascular stability, and other relevant indicators.
  • Effective Communication and Collaboration:
    • Foster effective communication and collaboration among healthcare team members, ensuring seamless coordination in the assessment, diagnosis, and treatment of patients with suspected or confirmed pulmonary embolism.
  • Patient Education on Prevention:
    • Provide education to patients and caregivers about risk factors for pulmonary embolism and preventive measures, emphasizing early ambulation, hydration, and adherence to prescribed anticoagulant therapy where applicable.
  • Quality Improvement and Outcome Monitoring:
    • Implement quality improvement initiatives to enhance the identification and management of pulmonary embolism cases. Establish mechanisms for ongoing monitoring of patient outcomes and adherence to evidence-based practices.

 

Pathophysiology of Pulmonary Embolism (PE)

 

  • Thrombus Formation:
    • Pulmonary embolism often originates from deep vein thrombosis (DVT), where a blood clot forms in the deep veins of the lower extremities or pelvis. This thrombus can dislodge and travel through the bloodstream.
  • Embolization to Pulmonary Arteries:
    • The thrombus, now an embolus, can travel through the venous system, reaching the right side of the heart. From there, it can be propelled into the pulmonary arteries, causing a blockage.
  • Obstruction of Pulmonary Vasculature:
    • As the embolus lodges in the pulmonary arteries, it obstructs blood flow to a portion of the lung, leading to impaired gas exchange and increased pulmonary vascular resistance.
  • Pulmonary Infarction and Inflammation:
    • Severe cases of PE may result in pulmonary infarction, causing localized lung tissue damage. Additionally, the sudden obstruction triggers an inflammatory response, contributing to symptoms and complications.
  • Hemodynamic Consequences:
    • The obstruction of pulmonary blood flow increases right ventricular afterload, leading to right ventricular strain. In severe cases, this can progress to right heart failure, causing systemic hypotension and compromised cardiac output.

Etiology of Pulmonary Embolism (PE)

 

  • Deep Vein Thrombosis (DVT):
    • The most common cause of pulmonary embolism is the migration of a blood clot from a deep vein, typically in the legs or pelvis. Conditions promoting DVT include prolonged immobility, surgery, trauma, and certain medical conditions.
  • Hypercoagulable States:
    • Individuals with hypercoagulable conditions, such as inherited clotting disorders or acquired conditions like antiphospholipid syndrome, are at an increased risk of developing blood clots that may lead to pulmonary embolism.
  • Venous Stasis:
    • Conditions that promote venous stasis, such as prolonged bed rest, long flights, or congestive heart failure, contribute to the development of clots that can embolize to the pulmonary arteries.
  • Trauma or Surgery:
    • Surgical procedures, particularly orthopedic surgeries, can increase the risk of clot formation. Trauma, especially fractures or extensive soft tissue injuries, can also predispose individuals to PE.
  • Cancer and Chemotherapy:
    • Cancer, especially certain types like lung, pancreatic, or ovarian cancer, is associated with an increased risk of developing blood clots. Additionally, some chemotherapy agents can contribute to hypercoagulability.

 

Desired Outcome for Pulmonary Embolism Nursing Care

 

  • Effective Oxygenation:
    • Ensure adequate oxygenation and maintenance of optimal oxygen saturation levels to prevent hypoxia and support respiratory function.
  • Hemodynamic Stability:
    • Maintain hemodynamic stability by preventing further right ventricular strain, optimizing cardiac output, and preventing complications such as shock.
  • Pain Management:
    • Alleviate and manage pain associated with pulmonary embolism, promoting the patient’s comfort and facilitating participation in therapeutic activities.
  • Prevention of Complications:
    • Minimize the risk of complications such as deep vein thrombosis, recurrent embolism, and pulmonary infarction through timely and appropriate interventions.
  • Patient Education and Psychosocial Support:
    • Provide comprehensive patient education on anticoagulant therapy, signs of recurrence, and lifestyle modifications to prevent future clots. Offer psychosocial support to address emotional and psychological aspects of the diagnosis.

Subjective Data:

 

  • Reports sudden onset of chest pain, described as sharp or stabbing.
  • Complains of shortness of breath, especially upon exertion.
  • Describes a feeling of anxiety or impending doom.
  • Reports recent history of prolonged immobility, such as a long flight or bed rest.
  • Mentions a personal or family history of deep vein thrombosis (DVT).
  • Expresses awareness of risk factors, such as recent surgery or trauma.
  • Reports cough, possibly with hemoptysis (coughing up blood).

 

Objective Data:

 

  • Elevated respiratory rate (tachypnea).
  • Increased heart rate (tachycardia).
  • Decreased oxygen saturation levels.
  • Crackles or wheezing upon lung auscultation.
  • Presence of a pleuritic rub on chest examination.
  • Signs of deep vein thrombosis (DVT) if present, such as unilateral leg swelling and tenderness.
  • Abnormal findings on imaging studies (CT pulmonary angiography, ventilation-perfusion scan).
  • Signs of right ventricular strain on electrocardiogram (ECG).
  • Elevated D-dimer levels.
  • Hypotension or a drop in blood pressure.

 

Nursing Assessment for Pulmonary Embolism

 

  • Respiratory Assessment:
    • Monitor respiratory rate, depth, and pattern to identify signs of respiratory distress, such as increased respiratory rate, dyspnea, and use of accessory muscles.
  • Oxygenation Status:
    • Assess oxygen saturation levels using pulse oximetry. Maintain SaO2 within the target range and intervene promptly if levels fall below the acceptable range.
  • Cardiovascular Assessment:
    • Monitor vital signs regularly, paying close attention to heart rate, blood pressure, and signs of right ventricular strain. Assess for the presence of jugular venous distension and peripheral edema.
  • Pain Assessment:
    • Evaluate the patient’s pain intensity, location, and characteristics. Use a pain scale to assess and document pain regularly. Implement appropriate pain management strategies.
  • Neurological Assessment:
    • Monitor neurological status, including mental status, orientation, and response to stimuli. Note any changes in consciousness or neurological deficits that may indicate complications.
  • Laboratory Monitoring:
    • Regularly assess laboratory values, including D-dimer, to aid in the diagnosis and monitor the effectiveness of anticoagulant therapy. Monitor for signs of bleeding related to anticoagulation.
  • Mobility and Ambulation:
    • Encourage and assess the patient’s mobility, promoting early ambulation when appropriate to prevent venous stasis and enhance overall circulation.
  • Psychosocial Assessment:
    • Evaluate the patient’s emotional and psychological well-being, addressing anxiety, fear, and concerns related to the diagnosis of pulmonary embolism. Collaborate with other healthcare professionals for additional support.

Interventions and Rationales

 

  • Administer Oxygen Therapy:
    • Rationale: Increases oxygen levels, addressing hypoxemia associated with pulmonary embolism, and supports respiratory function.
  • Initiate Anticoagulant Therapy (e.g., Heparin):
    • Rationale: Prevents further clot formation and reduces the risk of additional emboli, promoting anticoagulation.
  • Administer Analgesics for Pain Management:
    • Rationale: Relieves chest pain, promoting comfort and reducing anxiety, which can improve respiratory effort.
  • Implement Bed Rest:
    • Rationale: Reduces oxygen demand and minimizes the risk of dislodging clots, preventing further complications.
  • Monitor Vital Signs Closely:
    • Rationale: Allows for early detection of changes in respiratory rate, heart rate, and blood pressure, providing prompt intervention if needed.
  • Assist with Diagnostic Tests (e.g., CT Angiography, D-dimer):
    • Rationale: Aids in confirming the diagnosis and determining the extent of pulmonary embolism for appropriate management.
  • Elevate Legs and Encourage Ambulation (when appropriate):
    • Rationale: Enhances venous return and reduces the risk of deep vein thrombosis (DVT), a common precursor to pulmonary embolism.
  • Provide Emotional Support and Education:
    • Rationale: Addresses anxiety and fear, enhances coping mechanisms, and educates the patient on medication adherence and lifestyle modifications.
  • Prepare for Surgical Intervention (Embolectomy or Vena Cava Filter Placement):
    • Rationale: In cases of severe or recurrent pulmonary embolism, surgical interventions may be necessary to remove or prevent further emboli.
  • Collaborate with Respiratory Therapy for Breathing Exercises:
    • Rationale: Promotes optimal lung function, assists in preventing atelectasis, and supports respiratory recovery.

 

Evaluation of Pulmonary Embolism Nursing Care

 

  • Resolution of Symptoms:
    • Evaluate the effectiveness of interventions by assessing the resolution of symptoms such as dyspnea, chest pain, and tachycardia. Document improvements or persistence of symptoms.
  • Stable Oxygenation:
    • Monitor and evaluate oxygen saturation levels, ensuring they remain within the target range. Evaluate the need for continued oxygen therapy and adjust accordingly.
  • Achievement of Therapeutic Anticoagulation:
    • Regularly assess laboratory values to ensure therapeutic anticoagulation. Evaluate the effectiveness of anticoagulant therapy in preventing further clot formation.
  • Effective Pain Management:
    • Assess the patient’s pain levels regularly and document changes. Evaluate the effectiveness of pain management strategies, adjusting the plan as needed to ensure optimal pain relief.
  • Prevention of Complications:
    • Assess for the absence or reduction of complications, such as bleeding or recurrent embolism. Evaluate the success of preventive measures and interventions implemented to minimize the risk of complications.

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Transcript

All right. Today, we are going to be talking about pulmonary embolism. Let’s take a look at the patho. Pretty much, Pulmonary embolism is your gas exchange as impaired because there’s a blockage in the lung. It can be from a blood clot, fat, or air, some things that we want to consider as nurses. So, first we want to look at all the vital signs. What are some vital signs that you think we would see? What are some changes? I definitely think that maybe we’ll have some increased respirations, Uh, we’ll have a heart rate increase, so it will be a little tachy, And also, we may have a fever, or some low BP. The next thing we want to consider is starting some anticoagulation therapy. So, we want to start anticoagulation therapy, or we want to start some thrombolytics. We want to monitor bleeding and we want to initiate bleeding precautions because of those thrombolytics and anticoagulants, and we want to make sure we educate the patient about bleeding risk, and some things that they can do at home. The most important thing that we want to focus on with PEs is we want to stop the PE from growing in order to restore lung perfusion. 

 

So this right here is Virchow’s triad, and it’s just a fancy way of saying that these three things together are going to increase a patient’s risk at having the thrombus. So, right here we have hypercoagulability, which is pretty much how thick the blood is. Venous Stasis is just what it sounds, It is the blood that is stopped. So, this can increase with people who are immobile, or if they’re obese or have a more sedintary lifestyle. And finally, the last portion of the Virchow’s triad is the, uh, damaged blood vessels, so, that comes from IV drug, user atherosclerosis. All of these things together create the perfect environment for a thrombus to form and break off into the lungs, and that’s when you get the PE. 

 

So some suggestive data, what is the patient going to tell you that they’re feeling, they may complain about some pleuritic chest pain, so they want to complain about some pain, or they may also talk about some difficulty breathing or dyspnea, so, um, they may complain about that. And then you may also notice that they complain about dizziness, weakness, those types of things. 

 

Some things that we may observe as nurses remember objective data, objective observed. We may observe some respiration, so their respirations are going to be way up over 20. Okay, they’re going to have some tachycardia, so they’re going to have increased heart rate. They may have a temperature, a fever that’s a hundred and four or greater. Um, we may have some bloody sputum, some hemoptysis, some crackles when we listen. Some wet lungs is what I call it. We may also have a cough, uh, decreased SATs, decreased o2 SATs, so that may be anywhere, um, in the eighties, anything less than eighty-eight, we definitely want to be concerned about. And then we’re going to have, uh, increased D dimer. Some nursing interventions, so we are going to focus on a few things. 

A lot of things that we’re going to focus on are going to work on the clot, but we’re going to also want to take a look. So, the first thing we’re going to say here is monitoring vital signs. So, we’re going to want to monitor vital signs. So again, you’re going to have some hypoxia. So, you’re going to have some increased heart rate, increased respirations. This is going to be low SATs, so we can maybe, uh, administer some supplemental oxygen, um, if their saturations are low. So, we want to administer o2. The next thing we want to focus on is we want to go ahead and start that anticoagulation therapy or a thrombolytic. So, anticoagulants such as heparin and then there’s also a Alteplase, which is the thrombolytic. Anticoagulation heparin is going to stop the clot from growing, Alteplase is going to actually bust that clot up. 

The next thing we want to focus on is, uh, we are going to actually place that patient on bleeding precautions. So, we want to make sure that we minimize our blood sticks. So, bleeding precautions, we’re going to, uh, minimize blood, uh, lab sticks. We’re going to ensure that they use electric trimmers, soft bristled toothbrushes, things like that. The labs that we’re going to look at, we’re going to focus primarily on lactic acid and D dimer. Um, and D dimer is just telling you that there’s a thrombus or something in the blood, um, and that lactic acid is going to show some hypoxia. Um, we’re going to order a CT scan. CT scan is the way to diagnose a PE. There’s also something called VQ perfusion scan. We’re not going to get into too much detail with that, but pretty much that’s just to let you know that there is a clot or thrombus somewhere in the body. So, um, but CT scan is the gold standard. 

 

Finally, we want to collect the ABG. That ABG is going to get us a lot of good information, uh, with the patient’s respiratory status. So ABG, remember, they have the increased respiration rate. So, that is going to, um, show maybe some alkalosis and then they may progress, uh, to, uh, acidosis just based off of, uh, prolonged hypoxia. Uh, there are just a couple things here on this slide that I want to let you know about, and this is the thrombolytic, the absolute contraindication, so if they have anything in the last few months, any trauma in the last couple of months in the, uh, active, uh, recent intracranial bleeding, any surgery, active, a neoplasm or some tumor or cancer in the cranium. And then also if they have a history of hemorrhagic stroke, we want to stay away from thrombolytics because that’s going to also increase their, uh, chance and risk of bleeding. 

Uh, finally, these are our key points. Know this, okay. know this, remember the patho. PE is a block in the lungs that’s keeping your air from getting into your blood. Okay. They may complain of some dyspnea, some difficulty breathing, chest pain. They’re going to have some increased respirations. They’re going to be tachycardia, they’re going to have some, uh, bloody sputum, hemoptysis, decrease, uh, o2 SATs. Remember, we’re going to start them on anticoagulation or thrombolytics right away. Remember, those anticoagulants are going to keep it from growing thrombolytics or clot busters as I like to call them are going to break up and dissolve that clot. And then finally, we’re going to place them on bleeding precautions, because they are at increased risk of bleeding because of the medications that we’ve given them. We’re going to use a soft-bristle toothbrush, and we’re also going to advise them to follow these measures at home as long as they are on those anticoagulants. So we love you guys here. Go out and be your best self today and as always, happy nursing.

 

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