Nursing Care and Pathophysiology for Hypovolemic Shock

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Nichole Weaver
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Study Tools For Nursing Care and Pathophysiology for Hypovolemic Shock

Shock (Cheatsheet)
Rapid Infusion Hypovolemic Shock (Image)
Types of Shock (Picmonic)
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Outline

Pathophysiology: There is low blood flow either from hemorrhage, traumatic injury, dehydration, or burns. There is a loss of the circulating volume so there isn’t enough blood to enter the heart (preload), which decreases stroke volume and low cardiac output. The body will vasoconstrict to compensate. The body will shunt blood away from nonvital organs to vital organs.

Overview

  1. Hypovolemic Shock – loss of blood volume leading to decreased oxygenation of vital organs
  2. Body’s compensatory mechanisms fail and organs begin to shut down.

Nursing Points

General

  1. Stages of Hypovolemia
    1. Stage I – 500-750 mL Loss
    2. Stage II – 750 – 1500 mL Loss
    3. Stage III – 1500 – 2000 mL Loss
    4. Stage IV – > 2000 mL Loss

Assessment

  1. Symptoms
    1. Stage I – compensation
      1. May be asymptomatic
      2. Mild weakness
      3. Maybe pale
    2. Stage II – over compensation
      1. HR >100
      2. Pale
      3. Anxious
      4. UOP < 30mL/hr
    3. Stage III – failing compensation
      1. HR > 120
      2. ↓ BP
      3. ↓ LOC
      4. Pale, cool, clammy
      5. UOP <20 mL/hr
    4. Stage IV – failed compensation
      1. HR > 140
      2. SBP < 90
      3. RR > 30
      4. ↓ LOC → coma
      5. Weak pulse
      6. VERY pale, cool, diaphoretic
      7. No UOP
  2. Identify Cause
    1. Vomiting / diarrhea x days
    2. Severe burns
    3. Traumatic injury
    4. Hemorrhage (surgical, obstetric)

Therapeutic Management

  1. Treat Cause
    1. OR for repair
    2. Meds for vomiting / diarrhea
  2. Replace Volume
    1. Crystalloid – LR, NS
    2. Colloid – Blood Products
    3. Rapid Infuser
  3. Support Perfusion
    1. Hemodynamic Monitoring
    2. Vasopressors
  4. Life Support
    1. Decreased LOC = may need airway protection & ventilation

Nursing Care

  1. Fluid & Electrolytes
    1. 2 Large-bore IV’s
    2. Replace Volume IV
    3. RAPID IV Bolus
  2. Perfusion
    1. Monitor VS
    2. Hemodynamic Monitoring
      1. A-line
      2. Central Line
    3. Titrate Vasopressors
  3. Oxygenation
    1. Monitor airway/breathing
    2. Monitor SpO2 and LOC
    3. Give O2 as needed
    4. Prep for Life Support

Patient Education

  1. Reason for IV fluids
  2. Keep informed during treatment
  3. Possible informed consent for OR
  4. Blood transfusions

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Transcript

Okay guys, we’re gonna talk specifically about hypovolemic shock.

In hypovolemic shock, the initial insult, or the reason the organs aren’t receiving oxygen, is low blood volume. This could be because of a traumatic injury or hemorrhage, severe dehydration, or even burns can cause significant loss of circulating volume. If you guys can get the patho behind this, it will be easy to understand the symptoms. So if this is our circulatory system and this tank is our total blood volume, you can see here that the tank is low. So what happens is the blood that enters and fills the heart is decreased. Remember that’s called our preload. When our preload decreases, cardiac output also decreases and our body has mechanisms it uses to try to compensate. So you’ll see a lot of vasoconstriction in the body because it’s trying to push the blood back toward the heart – that means the pressure our heart has to pump against is increased – that’s our afterload. But it also means blood flow has shunted away from the non-vital organs like the skin – that’s why they get pale and cold. And then the heart rate will also increase to compensate for the lower stroke volume. After a while, we’ll begin to see our Blood pressure decrease because the body can only compensate for so long. Ultimately, there’s just not enough circulating blood volume to serve the whole system, and it will start to shut down.

So in our patients, the severity of the symptoms we see depends entirely on the amount of volume they’ve lost. In the first couple stages, they’ve lost a good bit of volume, but not so much that the body can’t compensate. You may not see any symptoms, but if you do they’re minor. You might see an elevated heart rate, they might be a little pale or cold, and you may start to see a decreased urine output because the kidneys are working to compensate by retaining fluid.

In stage III, this is where shock is kicking in. These compensatory mechanisms are failing and the patient is definitely in trouble. You’ll see their blood pressure decrease, their heart rate increases, and their urine output will be low for sure. They’ll be pale, clammy, and weak and will likely have a decreased level of consciousness because of the lack of oxygen to the brain. So we’re seeing this lack of perfusion to the kidneys and lack of perfusion to the brain – that’s shock. This patient needs intervention as soon as possible.

Stage IV is a dire life-threatening condition for the patient. Their blood pressure will be significantly low, their heart rate and respiratory rate will be significantly high. Their level of consciousness will be decreased – possibly to the point of them being unresponsive. Pulses may be thready, they will be very pale, cool, and clammy. And they will likely have little to no urine output at this point because of the lack of kidney flow. Now, you’ll notice that stage IV is a fluid loss of more than 2 L. The body only holds about 5 L of blood, so this person has lost 40% of their circulating volume! If we don’t restore the flow of oxygenated blood, this patient’s not gonna make it.

You know, sometimes you’ll see these things and you won’t know what’s really going on because there’s no obvious sign of bleeding or anything. This actually happened to me once in a post-op patient. They seemed fine, vitals were stable. They were complaining of being weak and they were a little pale, but I thought it was the anesthesia. They started to get anxious and their heart rate went up a little – but they were also in pain, so I just gave them some morphine. Next thing I knew, their heart rate was in the 120s, their blood pressure had dropped, and they were super confused. They had already pushed into Stage III and were showing signs of shock because their brain wasn’t getting enough oxygen. I called the surgeon and within 30 minutes my patient was back in the OR repairing a bleed. So it’s so important that you know when your patient is at risk and recognize these early signs for what they are! We want you to catch it sooner rather than later!

So what’s the treatment plan for a patient in hypovolemic shock? Well we ALWAYS want to treat the cause, whatever it was. But either way, we need to replace the volume they lost. This may be crystalloids or colloids, depending on the problem. Now, sometimes we just can’t replace fluids fast enough to support their blood pressure, so we may give things like vasopressors while we work on replacing volume. But it’s so important to remember that squeezing the tank does nothing if it’s empty. So always start filling the tank first. Also since these patients are at risk for a decreased LOC, they may actually need to be put on life support to protect their airway, depending on their situation.

When it comes to nursing care, there are a lot of priorities for this patient – one of which being that they need to be in an ICU. But we’re gonna focus on the top 3 concepts here. If you check out the outline and the care plan attached to this lesson, you’ll see a ton of details about specific interventions. The first concept is fluid & electrolytes because the #1 priority here is going to be to replace the volume they lost as fast as possible! When it comes to perfusion, we need to work to monitor and maintain their blood pressure. Then finally, these patients are definitely at risk for airway and breathing issues, so we need to monitor their oxygen status and intervene if needed.

So remember in hypovolemic shock, the initial insult is low blood volume – that causes a decrease in the oxygen being provided to the tissues. After a large amount of volume loss, the body loses its ability to compensate, hence the progression into shock. We want to treat the cause and we want to replace the volume they’ve lost (and then some). We will need to support their perfusion during this process so we’ll monitor them closely and possibly start vasopressors if necessary – just remember to fill the tank first! Keep your priority nursing concepts in mind and intervene as needed. And of course remember that this is an emergency. Recognize it and act quickly!

We want you guys to have peace of mind and be confident knowing that you can recognize this and even save a life! We love you guys, go out and be your best self today! And, as always, Happy Nursing!

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4th Semester

Concepts Covered:

  • Renal Disorders
  • Endocrine and Metabolic Disorders
  • Urinary System
  • Shock
  • Musculoskeletal Trauma
  • Postoperative Nursing
  • Preoperative Nursing
  • Cardiac Disorders
  • Renal and Urinary Disorders
  • Cardiovascular Disorders
  • Circulatory System
  • Respiratory System
  • Digestive System
  • Integumentary Disorders
  • Nervous System
  • Pregnancy Risks
  • Neurological Trauma
  • Neurologic and Cognitive Disorders
  • Emergency Care of the Neurological Patient
  • Neurological Emergencies
  • Respiratory Disorders
  • Substance Abuse Disorders
  • Central Nervous System Disorders – Brain
  • Basics of Sociology
  • Statistics
  • Urinary Disorders
  • Fundamentals of Emergency Nursing
  • Prioritization
  • Test Taking Strategies
  • Delegation
  • Documentation and Communication
  • Legal and Ethical Issues
  • Community Health Overview
  • Communication
  • Eating Disorders
  • Noninfectious Respiratory Disorder
  • Integumentary Disorders
  • Disorders of Pancreas
  • Upper GI Disorders
  • Acute & Chronic Renal Disorders
  • Liver & Gallbladder Disorders
  • Respiratory Emergencies
  • Emergency Care of the Cardiac Patient
  • Disorders of the Posterior Pituitary Gland

Study Plan Lessons

Fluid Volume Overload
Fluid Volume Deficit
Nursing Care and Pathophysiology for Sepsis
Nursing Care and Pathophysiology for SIRS & MODS
Nursing Care and Pathophysiology for Compartment Syndrome
Nursing Care and Pathophysiology for Rhabdomyolysis
Discharge (DC) Teaching After Surgery
Informed Consent
Performing Cardiac (Heart) Monitoring
Nephrotic Syndrome
Congenital Heart Defects (CHD)
EKG (ECG) Waveforms
The EKG (ECG) Graph
Electrical A&P of the Heart
Electrolytes Involved in Cardiac (Heart) Conduction
Breathing Movements
Breathing Control
Respiratory Functions of Blood
Liver & Gallbladder
Respiratory Structure & Function
Burn Injuries
Spinal Cord
Electrical Activity in the Heart
Cardiac (Heart) Physiology
Nutrition (Diet) in Disease
Blood Cultures
Drawing Blood
Spinal Precautions & Log Rolling
Neuro Assessment
Ischemic (CVA) Stroke Labs
Renal (Kidney) Failure Labs
Sepsis Labs
Dysrhythmias Labs
Anion Gap
Glucose Lab Values
Urinalysis (UA)
Glomerular Filtration Rate (GFR)
Creatinine (Cr) Lab Values
Blood Urea Nitrogen (BUN) Lab Values
Liver Function Tests
Total Bilirubin (T. Billi) Lab Values
Albumin Lab Values
Cultures
White Blood Cell (WBC) Lab Values
Hematocrit (Hct) Lab Values
Hemoglobin (Hbg) Lab Values
Red Blood Cell (RBC) Lab Values
Lab Panels
Urinary Elimination
Shock
Triage
Prioritization
Delegation
Documentation Pro Tips
Admissions, Discharges, and Transfers
Legal Considerations
Levels of Prevention
Nursing Care Delivery Models
Advance Directives
What Guides Nurses Practice
Fluid Compartments
Fluid Shifts (Ascites) (Pleural Effusion)
Phosphorus-Phos
ABGs Nursing Normal Lab Values
ABG (Arterial Blood Gas) Interpretation-The Basics
ROME – ABG (Arterial Blood Gas) Interpretation
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
Lactic Acid
Base Excess & Deficit
Burn Injuries
Nursing Care and Pathophysiology of Diabetic Ketoacidosis (DKA)
Nursing Care and Pathophysiology for Pancreatitis
Nursing Care and Pathophysiology of Acute Kidney (Renal) Injury (AKI)
Chronic Renal (Kidney) Module Intro
Nursing Care and Pathophysiology of Chronic Kidney (Renal) Disease (CKD)
Nursing Care and Pathophysiology for Cholecystitis
Nursing Care and Pathophysiology for Hepatitis (Liver Disease)
Nursing Care and Pathophysiology for Cirrhosis (Liver Disease, Hepatic encephalopathy, Portal Hypertension, Esophageal Varices)
Restrictive Lung Diseases (Pulmonary Fibrosis, Neuromuscular Disorders)
Nursing Care and Pathophysiology of Acute Respiratory Distress Syndrome (ARDS)
Blunt Chest Trauma
Nursing Care and Pathophysiology for Pneumothorax & Hemothorax
Brain Death v. Comatose
Nursing Care and Pathophysiology for Parkinsons
Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)
Nursing Care and Pathophysiology for Ischemic Stroke (CVA)
Stroke Assessment (CVA)
Stroke Therapeutic Management (CVA)
Stroke Nursing Care (CVA)
Seizures Module Intro
Spinal Cord Injury
Preload and Afterload
Nursing Care and Pathophysiology of Angina
Heart (Cardiac) Failure Module Intro
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Atrial Flutter
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Legal Aspects of Documentation
Dehydration
Cerebral Palsy (CP)
Spina Bifida – Neural Tube Defect (NTD)
Vasopressin
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)