Electrical A&P of the Heart

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Outline

Electrical A&P of the Heart Overview

  1. A&P of the heart’s electrical conduction and how it stimulates the atria and ventricles to contract
  2. Heart has 4 chambers, 2 upper (Atria) and 2 lower (ventricles)

Nursing Points for Electrical A&P of the Heart

General

  1. Cardiac cycle
    1. Atria contract (depolarize)  
      1. Pump blood to ventricles
    2. Ventricles contract (depolarize)
      1. Pump blood to body/lungs
    3. Atria and Ventricles rest (repolarize)
      1. Fill with blood
    4. Creates PQRST waveforms
  2. SA node (Sinoatrial node)
    1. Natural pacemaker of heart
    2. Electrical stimulus sent to R  & L atria
      1. Initiates a contraction
    3. Signals at 60-100 bpm
  3. AV node (Atrioventricular node)
    1. Slows down the  electrical signal from SA node
    2. Sends electrical impulse to the ventricles
    3. Intrinsic rate of  40-60 bpm
  4. Bundle of His
    1. Transmits electrical impulse from AV node to the ventricles
    2. Separates into Rt & Lt  bundle branches
  5. Right and Left bundle branches (RBB & LBB)
    1. RBB sends electrical impulse to Rt  ventricle
      1. Ventricle contracts
    2. LBB sends electrical impulse to Lt ventricle
      1. Ventricle contracts
    3. Intrinsic rate of 20-40 bpm
  6. Purkinje Fibers
    1. Last part of the electrical stimulus
    2. Sends impulse to the myocardium

Assessment of Electrical A&P of the Heart

  1. Apical pulse for 1 minute
  2. Recognize PQRST waveforms on ECG.

Nursing Concepts for Electrical A&P of the Heart

  1. EKG Rhythms

Patient Education

  1. Electrodes will be placed on the chest
    1. Do not apply lotion to chest
    2. May need to shave area where electrodes will be placed on skin

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Transcript

Hello, today we are going to talk about the electrical anatomy and physiology of the heart.

Okay let’s start by first reviewing the heart, remember that our heart has four chambers two upper and two lower. the two upper chambers are called the Atria, we have a right and left Atria. The two lower chambers are called the ventricles, we have a right and the left ventricle. It is important to understand that the Atria pump blood into the ventricles, the right ventricle pumps blood to the lungs and the left ventricle pumps blood to the body. In order for the Atria and the ventricles to contract there must be an electrical stimulation. Without an electrical stimulation the heart will not pump effectively. When the heart produces an electrical conduction it causes a mechanical action, meaning the Atria and the ventricles contract.

Mechanical action is simply referring to the heart’s ability to contract. In order for the heart to contract it goes through a cycle (Cardiac Cycle). The cycle starts when the atria receive an electrical stimulus causing the atria to contract. As the atria are contracting they are filling up the ventricles with blood, so the ventricles are in a resting state. When the atria are done filling the ventricles up with blood, they are in a resting state while the ventricles contract. If the Atria are contracting the ventricles are resting , if the ventricles are contracting the atria is resting. The term depolarize and repolarize simply mean contract and relax or resting state. You will hear depolarize and repolarize used more often versus contract or relax. So when the atria are depolarizing the are contracting, and the ventricles are repolarizing or resting. When the ventricles are depolarizing they are contracting and the atria are repolarizing. If you can remember that” re”polarizing means to” re”st or” re”-energize you will know that is the period where the atria or ventricles are resting, and depolarizing when contracting. The Mechanical action of Contracting or depolarizing and resting or repolarizing formulate the waveforms on an EKG

The mechanical action would not be possible without an electrical conduction. This is a quick overview of the electrical pathway that starts with the SA node followed by the AV node then down the bundle of His which separates into the right and left bundle branches and lastly the purkinje fibers. These look at these in more detail in the following slides.

The first step in the electrical conduction is the firing or the electrical stimulation of the SA node. The SA node is referred to as the natural pacemaker of the heart because it is what initiates the electrical conduction. The SA node sends an electrical impulse across the right and left Atria, this causes the atria to contract. The SA node sends an electrical impulse of 60 to 100 beats per minute across the atria. One cool thing to remember about the SA node is that it does not require a signal or stimulus to initiate an electrical impulse, it does it all on its own. The heart does not need the brain or any other organ in order to beat, the SA node will automatically send an electrical impulse to generate a contraction.

After the SA node sends its electrical impulse to the atria, the impulse travels to the AV node. The AV node is located between the Atria and ventricles which is why it’s also called atrioventricular node. The AV node slows down the electrical impulse from the SA node in order to make sure the Atria have contracted completely and the blood is expelled into the ventricles. When the Atria have finished contracting the AV node sends the electrical impulse down to the ventricles. If the SA node is not working or firing like it’s supposed to, the AV node can take over and produce a heart rate of 40-60 beats per minute.

After the electrical conduction passes through the AV node it goes through the bundle of His, which then separates into the right and left bundle branches. The right bundle branch sends its electrical impulse to the right ventricle and the left bundle branch sends its impulse to the left ventricle causing them to contract. Just like the AV node, if the SA node and the AV node were to malfunction the right and left bundle branches can pick up and produce a heart rate of 20-40 beats per minute, its the last effort before they die. A ventricular heart beat of 20-40 beats per minute would not perfuse the body well and the patient will die soon without an intervention like a pacemaker.

The last part of the electrical conduction is the purkinje fibers, These little electrical fibers send the electrical impulse to the ventricles and the rest of the myocardium to make sure the entire ventricle contracts. So without the initiation of the SA node firing across Across the internodal pathways in the atria, traveling to the AV node, down the bundle of His to the right and left bundle branches and ending at the purkinje fibers we would not have a mechanical action.

The key points to remember are that and electrical conduction stimulates the Atria and the ventricles to contract causing a mechanical action. The Atria and the ventricles contract or depolarize and relax or repolarize. When the Atria are depolarizing the ventricles are resting so they are repolarizing because they’re filling with blood while the Atria contract, when the Atria are done contracting it is their turn to repolarize while the ventricles depolarize or contract. Both atria and ventricles cannot be contracting at the same time or relaxing at the same time, your patient is dead! In order to have electrical stimulation it must follow a conduction pathway by starting at the SA node, and travel through the AV node bundle of His right and left bundle branches and purkinje fibers. When the atria and ventricles are depolarizing and repolarizing (contracting and relaxing) due to the electrical stimulation, waveforms on an EKG are being produced. These waveforms are discussed in the upcoming EKG lessons.

Check out our other resources and lessons on the electrical activity of the heart or any other topic you may need help with, as always, go out and be your best selves today and happy nursing!

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4th Semester

Concepts Covered:

  • Renal Disorders
  • Endocrine and Metabolic Disorders
  • Urinary System
  • Shock
  • Musculoskeletal Trauma
  • Postoperative Nursing
  • Preoperative Nursing
  • Cardiac Disorders
  • Renal and Urinary Disorders
  • Cardiovascular Disorders
  • Circulatory System
  • Respiratory System
  • Digestive System
  • Integumentary Disorders
  • Nervous System
  • Pregnancy Risks
  • Neurological Trauma
  • Neurologic and Cognitive Disorders
  • Emergency Care of the Neurological Patient
  • Neurological Emergencies
  • Respiratory Disorders
  • Substance Abuse Disorders
  • Central Nervous System Disorders – Brain
  • Basics of Sociology
  • Statistics
  • Urinary Disorders
  • Fundamentals of Emergency Nursing
  • Prioritization
  • Test Taking Strategies
  • Delegation
  • Documentation and Communication
  • Legal and Ethical Issues
  • Community Health Overview
  • Communication
  • Eating Disorders
  • Noninfectious Respiratory Disorder
  • Integumentary Disorders
  • Disorders of Pancreas
  • Upper GI Disorders
  • Acute & Chronic Renal Disorders
  • Liver & Gallbladder Disorders
  • Respiratory Emergencies
  • Emergency Care of the Cardiac Patient
  • Disorders of the Posterior Pituitary Gland

Study Plan Lessons

Fluid Volume Overload
Fluid Volume Deficit
Nursing Care and Pathophysiology for Sepsis
Nursing Care and Pathophysiology for SIRS & MODS
Nursing Care and Pathophysiology for Compartment Syndrome
Nursing Care and Pathophysiology for Rhabdomyolysis
Discharge (DC) Teaching After Surgery
Informed Consent
Performing Cardiac (Heart) Monitoring
Nephrotic Syndrome
Congenital Heart Defects (CHD)
EKG (ECG) Waveforms
The EKG (ECG) Graph
Electrical A&P of the Heart
Electrolytes Involved in Cardiac (Heart) Conduction
Breathing Movements
Breathing Control
Respiratory Functions of Blood
Liver & Gallbladder
Respiratory Structure & Function
Burn Injuries
Spinal Cord
Electrical Activity in the Heart
Cardiac (Heart) Physiology
Nutrition (Diet) in Disease
Blood Cultures
Drawing Blood
Spinal Precautions & Log Rolling
Neuro Assessment
Ischemic (CVA) Stroke Labs
Renal (Kidney) Failure Labs
Sepsis Labs
Dysrhythmias Labs
Anion Gap
Glucose Lab Values
Urinalysis (UA)
Glomerular Filtration Rate (GFR)
Creatinine (Cr) Lab Values
Blood Urea Nitrogen (BUN) Lab Values
Liver Function Tests
Total Bilirubin (T. Billi) Lab Values
Albumin Lab Values
Cultures
White Blood Cell (WBC) Lab Values
Hematocrit (Hct) Lab Values
Hemoglobin (Hbg) Lab Values
Red Blood Cell (RBC) Lab Values
Lab Panels
Urinary Elimination
Shock
Triage
Prioritization
Delegation
Documentation Pro Tips
Admissions, Discharges, and Transfers
Legal Considerations
Levels of Prevention
Nursing Care Delivery Models
Advance Directives
What Guides Nurses Practice
Fluid Compartments
Fluid Shifts (Ascites) (Pleural Effusion)
Phosphorus-Phos
ABGs Nursing Normal Lab Values
ABG (Arterial Blood Gas) Interpretation-The Basics
ROME – ABG (Arterial Blood Gas) Interpretation
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
Lactic Acid
Base Excess & Deficit
Burn Injuries
Nursing Care and Pathophysiology of Diabetic Ketoacidosis (DKA)
Nursing Care and Pathophysiology for Pancreatitis
Nursing Care and Pathophysiology of Acute Kidney (Renal) Injury (AKI)
Chronic Renal (Kidney) Module Intro
Nursing Care and Pathophysiology of Chronic Kidney (Renal) Disease (CKD)
Nursing Care and Pathophysiology for Cholecystitis
Nursing Care and Pathophysiology for Hepatitis (Liver Disease)
Nursing Care and Pathophysiology for Cirrhosis (Liver Disease, Hepatic encephalopathy, Portal Hypertension, Esophageal Varices)
Restrictive Lung Diseases (Pulmonary Fibrosis, Neuromuscular Disorders)
Nursing Care and Pathophysiology of Acute Respiratory Distress Syndrome (ARDS)
Blunt Chest Trauma
Nursing Care and Pathophysiology for Pneumothorax & Hemothorax
Brain Death v. Comatose
Nursing Care and Pathophysiology for Parkinsons
Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)
Nursing Care and Pathophysiology for Ischemic Stroke (CVA)
Stroke Assessment (CVA)
Stroke Therapeutic Management (CVA)
Stroke Nursing Care (CVA)
Seizures Module Intro
Spinal Cord Injury
Preload and Afterload
Nursing Care and Pathophysiology of Angina
Heart (Cardiac) Failure Module Intro
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Distributive Shock
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Atrial Flutter
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Legal Aspects of Documentation
Dehydration
Cerebral Palsy (CP)
Spina Bifida – Neural Tube Defect (NTD)
Vasopressin
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)