Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)

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Jon Haws
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Included In This Lesson

Study Tools For Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)

Hemorrhagic Stroke Risk Factors (Mnemonic)
Stroke Pathochart (Cheatsheet)
Intraparenchymal Hemorrhage (Image)
Cerebral Aneurysm (Image)
Coiled Aneurysm (Image)
Pureed Diet (Image)
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Outline

Pathophysiology: A vessel ruptures and bleeds into the brain. This puts pressure and blood on the brain as the blood accumulates. This can be caused by a weakened vessel such as in an aneurysm.

Overview

Lack of blood flow to brain tissue caused by bleeding in/around brain.

Nursing Points

General

  1. Pathophysiology
    1. Bleed in/around brain due to ruptured vessel
    2. Hypertension → weakened vessel
      1. i.e. aneurysm rupture
    3. No flow past point of bleed
    4. Visible immediately on CT scan
    5. Presents as “worst headache of my life” (especially Subarachnoid Hemorrhage)
  2. Risk Factors
    1. Hypertension
    2. Substance Abuse (cocaine)
    3. Anticoagulant Therapy
    4. Trauma
  3. Complications
    1. Blood = irritant to tissues
    2. Seizures
    3. Vasospasm – vessels clamp down
      1. Cause more ischemia

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Example Nursing Diagnosis For Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)

  1. Impaired Physical Mobility: Stroke often results in impaired mobility or paralysis. This diagnosis focuses on mobility issues.
  2. Altered Cerebral Perfusion: Stroke can lead to cerebral perfusion deficits. This diagnosis addresses the impact on brain circulation.
  3. Risk for Aspiration: Stroke patients may have swallowing difficulties, increasing the risk of aspiration. This diagnosis emphasizes aspiration prevention.

ADPIE Related Lessons

Transcript

So let’s look specifically at hemorrhagic stroke. We’re going to talk about the pathophysiology and major points, then we’ll talk about assessment, therapeutic management, and nursing care in a later lesson.

A hemorrhagic stroke is a lack of blood flow to the brain tissue caused specifically by a bleed somewhere in or around the brain. Typically this occurs because one of the blood vessels in the brain has ruptured. In the cardiac course we talk a lot about hypertension and how much it can weaken those blood vessels, same with aneurysms. You can have these weakened blood vessels and weakened outpouchings in the brain as well. When one of them ruptures, blood flow beyond that spot is severely diminished. No blood flow, remember, always leads to death of the tissue. It’s like trying to water your flowers when there’s a hole in the side of your hose. So not only do we lose blood flow, but now we start building up blood where it doesn’t belong – and if you remember from the ICP lesson, that’s going to increase our intracranial pressure. In addition to other neurological symptoms of stroke that we’ll look at in the assessment lesson, these patients often complain that this is the worst headache of their life, sometimes it even wakes them out of their sleep. When we do a CT scan, we will be able to see immediately that there is bleeding on the brain, like you can see here.

Risk factors for hemorrhagic strokes, again hypertension is a huge one as well as substance abuse, specifically cocaine use. Both hypertension and cocaine will weaken these vessel walls until they burst. We also need to consider anyone on anticoagulant therapy as being at risk – especially our little elderly patients who are on warfarin for their A-Fib, but also are losing their balance a lot – if they fall and hit their head, it could cause damage to the vessels and lead to a hemorrhagic stroke – especially because their body is not clotting like it should.

There are a couple of complications that are high-risk in a patient with a hemorrhagic stroke and they both relate to the fact that blood, when it is somewhere it’s not supposed to be, is very irritating. Keep that in mind for the whole body, not just the brain – blood is an irritant. Now, remember we have our brain tissue and it’s covered by the Pia mater. Then we have our skull which is lined by the dura mater. And in between we have the arachnoid layer. Underneath this, in the subarachnoid space, there are tons of nerve endings. This is also where the majority of our major blood vessels are within the skull. If you start to get blood in this space, it’s going to irritate those nerve endings and those blood vessels. So you can see seizures as well as vasospasm. Vasospasm is when the blood vessels in the brain spasm or clamp down. So now, not only do you have the issue of the bleed, but now you’re getting ischemia because the vessels have clamped down. And 3 days after the stroke, you’ll suddenly see the patient develop new stroke symptoms. So you’ll see in the therapeutic management lesson the things that we do to mitigate these risks.

So just to recap, a hemorrhagic stroke is a lack of blood flow to the brain due to bleeding. Some modifiable risk factors are hypertension and substance abuse because of their effect on weakening the blood vessels. We need to be cautious with patients who are on anticoagulants, especially the elderly who are prone to Falls. And we need to take precautions to prevent complications like seizures and vasospasm.

Make sure you check out the rest of this module to learn more about how we manage stroke patients. In the nursing care lesson you’ll find a detailed care plan as well as a case study, so be sure to check that out. Now go out and be your best selves today. And, as always, happy nursing!

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Study Plan Lessons

Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Acute Kidney (Renal) Injury (AKI)
Nursing Care and Pathophysiology of Diabetes Mellitus (DM)
Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)
Nursing Care and Pathophysiology of COPD (Chronic Obstructive Pulmonary Disease)
Asthma
Nursing Care and Pathophysiology for Anemia
Fractures
Respiratory Acidosis (interpretation and nursing interventions)
ABGs Tic-Tac-Toe interpretation Method
ROME – ABG (Arterial Blood Gas) Interpretation
ABG (Arterial Blood Gas) Interpretation-The Basics
ABGs Nursing Normal Lab Values
ABG Course (Arterial Blood Gas) Introduction
Respiratory Alkalosis
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
ABG (Arterial Blood Gas) Oxygenation
Lactic Acid
Base Excess & Deficit
02.01 Hypertensive Crisis for CCRN Review
02.02 Cardiomyopathy for CCRN Review
02.06 Heart Murmurs for CCRN Review
02.07 Reading “A, C, V Waves” & PAWP Waveforms for CCRN Review
02.08 Cardiac Catheterization & Acute Coronary Syndrome for CCRN Review
02.09 12 Lead EKG- Leads 1, 2, 3, aVL, and aVF for CCRN Review
02.10 12 Lead EKG- Lead V1-V6 for CCRN Review
02.11 12 Lead EKG- Injuries for CCRN Review
02.12 Myocardial Infarction- Inferior Wall for CCRN Review
02.13 Myocardial Infarction – Anterior Septal Wall for CCRN Review
02.14 Shock Stages for CCRN Review
02.15 Hypovolemic Shock for CCRN Review
02.16 Cardiogenic Shock for CCRN Review
02.17 Septic Shock for CCRN Review
02.18 Cardiovascular Practice Questions for CCRN Review
03.01 Syndrome of Inappropriate Antidiuretic hormone (SIADH) for CCRN Review
03.02 Diabetes Insipidus for CCRN Review
03.03 Hypoglycemia for CCRN Review
03.04 DKA vs HHNK for CCRN Review
03.05 Endocrine Practice Questions for CCRN Review
04.01 Hematology for CCRN Review
08.01 Psychological Review for CCRN Review
04.02 Hematology Review Questions for CCRN Review
05.01 Pancreatitis and Large Bowel Obstruction for CCRN Review
05.02 Liver Overview and Disease for CCRN Review
05.03 Jaundice for CCRN Review
05.04 Ruptured Spleen for CCRN Review
05.05 GI Practice Questions for CCRN Review
06.01 Organ Failure, Dysfunction & Trauma for CCRN Review
06.02 Poisoning for CCRN Review
06.03 Multi-System CCRN Important Points for CCRN Review
06.04 Differentiating Ectopy and Aberrancy for CCRN Review
06.05 Wide Complex Tachycardia for CCRN Review
07.01 CVA (Cerebrovascular Accident/Stroke) for CCRN Review
07.02 Neuro Anatomy for CCRN Review
07.03 Uncal Herniation for CCRN Review
07.04 Supratentorial Herniation and Glasgow Coma Scale for CCRN Review
07.05 Supratentorial Herniation: Cushings Triad for CCRN Review
07.06 Increased Intracranial Pressure (ICP) for CCRN Review
07.07 Cerebral Perfusion Pressure for CCRN Review
07.08 Basilar Skull Fracture for CCRN Review
07.09 Meningitis for CCRN Review
07.10 Neurologic Review questions for CCRN Review
09.01 Acute Renal Failure Overview for CCRN Review
09.02 Acute Tubular Necrosis for CCRN Review
09.03 Acute Renal (Pre-Renal vs Renal) Failure for CCRN Review
09.04 Continuous Renal Replacement Therapy for CCRN Review
09.05 Chronic Renal Failure for CCRN Review
09.06 Renal Practice Questions for CCRN Review
10.01 Arterial Blood Gas (ABG) Interpretation for CCRN Review
10.02 Breath Sounds for CCRN Review
10.03 Acute Respiratory Failure for CCRN Review
10.04 Pulmonary Question Review for CCRN Review
EKG (ECG) Course Introduction
Electrical A&P of the Heart
Electrolytes Involved in Cardiac (Heart) Conduction
The EKG (ECG) Graph
EKG (ECG) Waveforms
Calculating Heart Rate
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Atrial Flutter
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
Fluid & Electrolytes Course Introduction
Fluid Compartments
Fluid Pressures
Fluid Shifts (Ascites) (Pleural Effusion)
Isotonic Solutions (IV solutions)
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Potassium-K (Hyperkalemia, Hypokalemia)
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Chloride-Cl (Hyperchloremia, Hypochloremia)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Phosphorus-Phos
Blood Glucose Monitoring