1st Degree AV Heart Block

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Maria Stewart
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Included In This Lesson

Study Tools For 1st Degree AV Heart Block

Parts of EKG waveform (Image)
1st Degree AV Block (Image)
10 Common EKG Heart Rhythms (Cheatsheet)
EKG Chart (Cheatsheet)
EKG Electrical Activity Worksheet (Cheatsheet)
Heart Rhythms Signs and Symptoms (Cheatsheet)
Heart Rhythm Identification (Cheatsheet)
Heart Blocks (Cheatsheet)
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Outline

Overview

  1. 1st degree AV heart block
    1. Conduction delay in the AV node
      1. Prolonged conduction from the atria to ventricles
      2. PR interval >0.20 seconds

Nursing Points

General

  1. Characteristics of 1st degree AV heart block
    1. Rhythm
      1. Regular
      2. Irregular
    2. Rate
      1. Varies
      2. Depends on underlying rhythm
    3. P:QRS ratio
      1. 1:1
    4. PR interval
      1. >0.20 seconds
    5. QRS complex
      1. 0.06-0.12 second

Assessment

  1. Patient Presentation
    1. Asymptomatic
  2. VS

Therapeutic Management

  1. Nursing Interventions
    1. Continue to monitor
  2. Determine/treat possible causes
    1. Electrolytes
    2. Medications
    3. MI
  3. Symptomatic bradycardia
    1. ACLS guidelines

Nursing Concepts

  1. EKG Rhythms

Patient Education

  1. Count radial pulse
    1. Report if <60 or >100 and symptomatic

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Transcript

Hey guys in this lesson we are going to talk about first degree AV heart block, can also be called just 1st degree block. We are going to talk about the characteristics of it, and nursing interventions and treatments. So let’s get started.

So in first degree AV heart block the SA nodes initiates the impulse, it travels across the atria and then down to the AV node. Now remember the AV node naturally slows down the impulse so the atria fully contract. Well in 1st degree block the AV node will slow down the impulse too much so it takes longer than normal, so the signal from the atria to the ventricles takes longer. This is measured with the PR interval, remember that normally it takes 0.12-0.20 seconds. So in 1st degree block, the PR interval is greater 0.20 seconds, meaning it takes the electricity longer from the SA node to the ventricles because the AV node slows things down. So let’s break it down a little further.

So in step 1 we need to look at the rhythm and find out if it is a regular rhythm or an irregular rhythm. So we count the boxes in between the R waves and we have 27 consistently our rhythm is regular. Sometimes it can be irregular, again it depends on the underlying rhythm. An example would be if I have a sinus arrhythmia, it would be irregular. Ok so let’s do step 2 and count our heart rate, so we have 5 R waves and we multiply it by 10 since it is a 6 second strip and we get 50 beats per minute. In the 1500 method we divide 1500 by 27 and get 55 beats per minute which again is more accurate. So in step 3 we need to look at the P to QRS ratio, do we have 1 P wave followed by 1 QRS and we do, every P wave has a QRS behind it. In step 4 we look at the PR interval and we have 6 small boxes so our PR interval is 0.24 seconds. In step 5 we look at the QRS complex and we have about 1 and a half boxes so our QRS complex is 0.06 seconds. So step 6 would be to identify the rhythm and it is a Sinus bradycardia with a first-degree AV heart block. So always know the underlying rhythm. So let’s recap the characteristics, the rhythm is regular for the most part but it can be irregular depending on the underlying rhythm. The heart rate varies but usually with a first-degree AV block it’s a slower rate. There P:QRS ratio is 1 to 1. The PR interval is longer than 0.20 seconds and that is the biggest characteristic of 1st degree block and the QRS complex is normal. This is relatively a harmless rhythm unless the patient is really bradycardic and symptomatic. So let’s talk about managing it.

So usually when patients present with 1st degree AV block they are asymptomatic for the most part, may become symptomatic with bradycardia. Nursing interventions are to just continue to monitor them. If we can find out the cause we can try to treat it. Some of the possible causes are electrolyte imbalances or medications that slow down the AV node like calcium channel blockers or heart problems like an MI. Again this rhythm is harmless, if they become bradycardia and are symptomatic, we can follow ACLS guidelines for the most part we just continue to monitor the patients and rhythm.

So the key points that you need to remember from this lesson are the abnormalities of 1st degree AV heart blocks which like the name says, the AV node is slowing down the conduction and delays the impulse from the atria to the ventricles so the PR interval is going to be greater than 0.20 seconds. The nursing interventions are to just continue to monitor the patients, if they become symptomatic with bradycardia follow ACLS guidelines, if you can identify and treat the cause to improve 1st degree heart block
So I hope this little lesson makes it easier to understand 1st degree AV heart block. Make sure you check out all of the resources attached to this lesson. Now, go out and be your best self today! And, as always, happy nursing!

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Study Plan Lessons

Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Acute Kidney (Renal) Injury (AKI)
Nursing Care and Pathophysiology of Diabetes Mellitus (DM)
Nursing Care and Pathophysiology for Hemorrhagic Stroke (CVA)
Nursing Care and Pathophysiology of COPD (Chronic Obstructive Pulmonary Disease)
Asthma
Nursing Care and Pathophysiology for Anemia
Fractures
Respiratory Acidosis (interpretation and nursing interventions)
ABGs Tic-Tac-Toe interpretation Method
ROME – ABG (Arterial Blood Gas) Interpretation
ABG (Arterial Blood Gas) Interpretation-The Basics
ABGs Nursing Normal Lab Values
ABG Course (Arterial Blood Gas) Introduction
Respiratory Alkalosis
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
ABG (Arterial Blood Gas) Oxygenation
Lactic Acid
Base Excess & Deficit
02.01 Hypertensive Crisis for CCRN Review
02.02 Cardiomyopathy for CCRN Review
02.06 Heart Murmurs for CCRN Review
02.07 Reading “A, C, V Waves” & PAWP Waveforms for CCRN Review
02.08 Cardiac Catheterization & Acute Coronary Syndrome for CCRN Review
02.09 12 Lead EKG- Leads 1, 2, 3, aVL, and aVF for CCRN Review
02.10 12 Lead EKG- Lead V1-V6 for CCRN Review
02.11 12 Lead EKG- Injuries for CCRN Review
02.12 Myocardial Infarction- Inferior Wall for CCRN Review
02.13 Myocardial Infarction – Anterior Septal Wall for CCRN Review
02.14 Shock Stages for CCRN Review
02.15 Hypovolemic Shock for CCRN Review
02.16 Cardiogenic Shock for CCRN Review
02.17 Septic Shock for CCRN Review
02.18 Cardiovascular Practice Questions for CCRN Review
03.01 Syndrome of Inappropriate Antidiuretic hormone (SIADH) for CCRN Review
03.02 Diabetes Insipidus for CCRN Review
03.03 Hypoglycemia for CCRN Review
03.04 DKA vs HHNK for CCRN Review
03.05 Endocrine Practice Questions for CCRN Review
04.01 Hematology for CCRN Review
08.01 Psychological Review for CCRN Review
04.02 Hematology Review Questions for CCRN Review
05.01 Pancreatitis and Large Bowel Obstruction for CCRN Review
05.02 Liver Overview and Disease for CCRN Review
05.03 Jaundice for CCRN Review
05.04 Ruptured Spleen for CCRN Review
05.05 GI Practice Questions for CCRN Review
06.01 Organ Failure, Dysfunction & Trauma for CCRN Review
06.02 Poisoning for CCRN Review
06.03 Multi-System CCRN Important Points for CCRN Review
06.04 Differentiating Ectopy and Aberrancy for CCRN Review
06.05 Wide Complex Tachycardia for CCRN Review
07.01 CVA (Cerebrovascular Accident/Stroke) for CCRN Review
07.02 Neuro Anatomy for CCRN Review
07.03 Uncal Herniation for CCRN Review
07.04 Supratentorial Herniation and Glasgow Coma Scale for CCRN Review
07.05 Supratentorial Herniation: Cushings Triad for CCRN Review
07.06 Increased Intracranial Pressure (ICP) for CCRN Review
07.07 Cerebral Perfusion Pressure for CCRN Review
07.08 Basilar Skull Fracture for CCRN Review
07.09 Meningitis for CCRN Review
07.10 Neurologic Review questions for CCRN Review
09.01 Acute Renal Failure Overview for CCRN Review
09.02 Acute Tubular Necrosis for CCRN Review
09.03 Acute Renal (Pre-Renal vs Renal) Failure for CCRN Review
09.04 Continuous Renal Replacement Therapy for CCRN Review
09.05 Chronic Renal Failure for CCRN Review
09.06 Renal Practice Questions for CCRN Review
10.01 Arterial Blood Gas (ABG) Interpretation for CCRN Review
10.02 Breath Sounds for CCRN Review
10.03 Acute Respiratory Failure for CCRN Review
10.04 Pulmonary Question Review for CCRN Review
EKG (ECG) Course Introduction
Electrical A&P of the Heart
Electrolytes Involved in Cardiac (Heart) Conduction
The EKG (ECG) Graph
EKG (ECG) Waveforms
Calculating Heart Rate
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Atrial Flutter
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
Fluid & Electrolytes Course Introduction
Fluid Compartments
Fluid Pressures
Fluid Shifts (Ascites) (Pleural Effusion)
Isotonic Solutions (IV solutions)
Hypotonic Solutions (IV solutions)
Hypertonic Solutions (IV solutions)
Potassium-K (Hyperkalemia, Hypokalemia)
Sodium-Na (Hypernatremia, Hyponatremia)
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Chloride-Cl (Hyperchloremia, Hypochloremia)
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Phosphorus-Phos
Blood Glucose Monitoring