Nursing Care and Pathophysiology for Heart Failure (CHF)

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Nichole Weaver
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Included In This Lesson

Study Tools For Nursing Care and Pathophysiology for Heart Failure (CHF)

Heart Failure-Origin (Mnemonic)
Heart Failure – Right Sided (Mnemonic)
Heart Failure-Left-Sided (Mnemonic)
Heart Failure Pathochart (Cheatsheet)
R v L Heart Failure (Cheatsheet)
Pitting Edema (Image)
Jugular Venous Distension (Image)
Heart Failure CHF Cardiac (Image)
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Outline

 

Pathophysiology: In heart failure, the heart does not pump effectively. This can occur because of many reasons but usually, because there has been damage to the heart tissue. The heart is not able to pump enough fluid forward so fluid then backs up. This fluid backup increases work on the heart as it tries to keep up and cannot.


Overview

The heart is a pump, circulates blood throughout the body. Heart failure = pump failure. Heart failure occurs when the heart cannot pump enough blood to supply the body’s needs.


Nursing Points

General

  1. Pump Failure
    1. Decreased perfusion forwards
    2. Increased congestion backwards
  2. Causes
    1. Myocardial Infarction
      1. Dead muscle can’t pump
    2. Hypertension
      1. ↑ afterload = ↑ stress on heart muscle
    3. Valve Disorders
      1. Blood not moving in right direction
      2. Inefficient pump
  3. Diagnostics
    1. BNP (Brain Natriuretic Peptide) – stretch of LV
    2. Echocardiogram
      1. Ejection Fraction
      2. Can diagnose valve disorder
    3. Chest X-Ray (CXR
      1. Cardiomegaly
      2. Pulmonary Edema
  4. Complications
    1. Volume Overload
      1. Pulmonary Edema
      2. Exacerbations
    2. Decreased Perfusion
      1. Heart
        1. Angina, MI
        2. Arrhythmias
      2. Organs
        1. Impaired Kidney Function

Assessment

  1. Right-Sided Heart Failure
    1. Decreased Pulmonary Perfusion
      1. ↓ oxygenation
      2. ↓ activity tolerance
    2. Increased Systematic Congestion
      1. Peripheral Edema
      2. ↑ JVD
      3. ↑ Preload
      4. Weight Gain
      5. Fatigue
      6. Liver / GI Congestion
  2. Left Sided Heart Failure
    1. Decreased Systemic Perfusion
      1. Skin pale or dusky
      2. ↓ Peripheral pulses
      3. Slow  capillary refill
      4. ↓ renal perfusion
        1. ↓ urine output
        2. Kidney Injury / Failure
    2. Increased Pulmonary Congestion
      1. Pulmonary edema
        1. Cough
        2. Pink/frothy sputum
        3. Crackles
        4. Wheezes
        5. Tachypnea
        6. SOB on Exertion
      2. Anxiety/restlessness

Therapeutic Management

Goal is to decrease workload on heart while still increasing cardiac output. Discussed in more detail in Therapeutic Management Lesson

  1. Decrease Preload
  2. Decrease Afterload
  3. Increase Contractility

Patient Education

Discussed in more detail in Therapeutic Management Lesson

  1. Diet & Lifestyle Changes
  2. Medication Instructions
  3. Activity Restrictions
  4. Frequent Follow-Ups

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ADPIE Related Lessons

Related Nursing Process (ADPIE) Lessons for Nursing Care and Pathophysiology for Heart Failure (CHF)

Transcript

This lesson is going to cover the basics of heart failure.

Heart disease is one of the leading causes of death in the US so you will see Heart Failure quite frequently in clinical practice. We want you to get an Ah-hah moment here and really understand this stuff. We’re gonna give you the most important things you need to know.

So, what exactly causes heart failure? Well remember it’s just pump failure – so anything that impacts the heart’s ability to pump efficiently can cause heart failure.

The top two causes are MI’s and hypertension, but valve disorders can cause a lot of problems, too. So what happens here? Well in an MI, if you remember from those lessons, blood supply is cut off to the heart muscle and the muscle itself dies. Muscle that is dead can’t contract, so the pumping ability or contractility of the heart is affected. Hypertension causes an increased afterload – remember from the hemodynamics lesson.

The heart has to pump harder against a higher pressure in the vessels. After a while, it can get worn out. Ultimately the ventricles can’t efficiently pump against that pressure any more. Someone with valve disorders, like stenosis or regurgitation, will have issues getting the blood where it’s supposed to be. If it’s not going where it’s supposed to, then it’s backing up – causing increased congestion or stretch or preload in the heart. This stress on the heart can lead to heart failure. So again, anything that impacts the heart’s ability to pump efficiently can cause heart failure – this means anything that affect the heart muscle.

BNP Heart Failure Guidelines

So there are multiple ways that we can diagnose and classify heart failure, but we just want you to know these three. The lab value we use is called Brain Natriuretic Peptide or BNP – this is released whenever the ventricles are stretched. So in Congestive Heart Failure patients when they’re severely volume overloaded, we can see this number jump into the thousands. In the labs course we will talk in much more detail about this value, so be sure to check that out.

Heart Failure Chest X-Ray

We’ll also do a chest x-ray, which could show a couple things. You may see the heart is actually enlarged because of the overload and stretch in the ventricles – and you may also see fluid in the lungs – that’s the pulmonary edema. We’ll also get an echocardiogram which can tell us things like end-diastolic volumes (remember that measures preload), ejection fraction, which is usually less than 50% in heart failure, and whether or not there are any valve issues.

Clinical Signs of Heart Failure

So let’s look at what this patient looks like when you actually see them in practice…

In the module intro we asked y’all to brainstorm what you thought this patient looked like. Guys if you get nothing else about Heart Failure, THIS is the part you’ve got to get.

Remember we said that there’s decreased perfusion forward and increased congestion backwards, right? So in right-sided heart failure we’re going to see some oxygenation problems because of decreased perfusion to the lungs. They may struggle with activity because they just don’t have enough blood flow for their lungs!

Then backwards we see that congestion into the system – they are way overloaded in the systemic circulation – so what does that mean? That means peripheral edema – okay how would you feel if you swelled up like crazy? You’d be tired, you’d probably gain some weight? Some of that fluid may even collect in the gut and make you nauseous.

Then because the blood can’t get past the heart from the body, it even backs up into the neck and you’ll see this crazy jugular venous distention – it looks like a rope on their neck!! So if you’re seeing these signs of excessive volume out in the body – you gotta think right sided heart failure!

Okay, so what about the left? Well they actually have decreased perfusion to the body. Remember your signs of decreased perfusion? They’re gonna be pale, decreased pulses, maybe slow cap refill, and their skin might even be cold, right? Signs of decreased perfusion. Then, we see the congestion happening in their lungs – y’all their lungs are full of fluid! How would you feel? You’d be struggling to breathe, right? They’ll have a cough, and their sputum will be pink and frothy because there’s just so much blood pumping through the lungs! Imagine if this was you and had all this fluid in your lungs – what position would you want to be in? You lay down and you’ll feel like you’re drowning – a lot of these patients sleep with lots of pillows or even in a recliner. Some of them even lose weight because it becomes a choice to either eat or breathe! So if you see these severe respiratory issues and fluid on the lungs, you’ve got to think Left-sided heart failure!

So right sided the classic sign is the systemic overload and left-sided classic sign is pulmonary edema. If you get this you can pick out any issues they’ll have!

Complications of Heart Failure

Now, there are quite a few other complications of heart failure, but the one I really want y’all to understand is what happens when the kidneys don’t get perfused. So here’s our heart failure patient who has decreased perfusion forward and increased congestion backward. They’re volume overloaded, probably struggling to breathe, and now, they aren’t perfusing their kidneys.

When the kidneys lose blood flow, it stimulates the Renin-Angiotensin-Aldosterone system (RAAS). You will learn more about this in pharmacology, but what you need to know is that it causes three main things to happen in the body.

  1. The first is water retention because of aldosterone and ADH. The kidneys see a lack of flow and think they need to hold onto water! So it is increasing the preload (or stretch) on ventricles whose preload is already sky-high!
  2. The second is vasoconstriction. This is the body trying to pull blood towards the heart to increase the blood pressure – this increases afterload (the force the heart has to pump against) – in a patient whose heart is already struggling as it is!
  3. And the third is the RAAS activates the Sympathetic Nervous System – it’s basically telling the heart to work harder and faster – which it cannot do! The end result is MORE volume overload, MORE stress on the heart muscle, and a perpetuated cycle that never ends.

That’s why I call it the cycle of death. What you’ll see when we look at therapeutic management in the next lesson is that the majority of therapy is aimed at breaking this cycle.

So let’s recap – anything that can affect the heart’s ability to pump effectively can cause heart failure, including an MI, hypertension, and valve disorders. Right-sided heart failure presents with symptoms of decreased pulmonary perfusion and increased systemic congestion – so they’re swollen, gain weight, lots of edema. Left-sided is the opposite – decreased systemic perfusion and increased pulmonary congestion, so these patients are really struggling to breathe. We diagnose with a BNP, Chest X-ray, and Echocardiogram. And don’t forget that the impact this has on the kidneys can make the problem worse – we’ll talk about how we work to break that cycle of death for these patients.

I hope we’ve been able to clear this up for you a bit, like we said sometimes heart failure is overcomplicated! Our goal is to make it easier to understand to give you that peace of mind and confidence! So go out and be your absolute best self today and, as always, happy nursing!

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Concepts Covered:

  • Cardiac Disorders
  • Cardiovascular
  • Circulatory System
  • Emergency Care of the Cardiac Patient
  • Shock
  • Shock
  • Disorders of Pancreas
  • Endocrine
  • EENT Disorders
  • Adult
  • Medication Administration
  • Acute & Chronic Renal Disorders
  • Respiratory Emergencies
  • Newborn Complications
  • Disorders of the Adrenal Gland
  • Disorders of the Thyroid & Parathyroid Glands
  • Immunological Disorders
  • Musculoskeletal Trauma
  • Hematologic Disorders
  • Vascular Disorders
  • Microbiology
  • Respiratory Disorders
  • Depressive Disorders
  • Oncology Disorders
  • Noninfectious Respiratory Disorder
  • Nervous System
  • Central Nervous System Disorders – Brain
  • Substance Abuse Disorders
  • Intraoperative Nursing
  • Emergency Care of the Trauma Patient
  • Integumentary Disorders
  • Integumentary Disorders
  • Urinary System
  • Liver & Gallbladder Disorders
  • Eating Disorders
  • Pregnancy Risks
  • Cardiovascular Disorders
  • Terminology
  • Disorders of Thermoregulation
  • Basics of NCLEX
  • Multisystem
  • Upper GI Disorders
  • Studying
  • Neurological Emergencies
  • Postpartum Complications
  • Infectious Respiratory Disorder
  • Renal Disorders
  • Communication
  • Perioperative Nursing Roles
  • Oncologic Disorders
  • Musculoskeletal Disorders
  • Prenatal Concepts
  • Muscular System
  • Proteins
  • Sexually Transmitted Infections
  • Peripheral Nervous System Disorders
  • Disorders of the Posterior Pituitary Gland

Study Plan Lessons

02.02 Cardiomyopathy for CCRN Review
02.03 Swan-Ganz Catheters for CCRN Review
02.04 Pulmonary Artery Wedge Pressure (PAWP) for CCRN Review
02.08 Cardiac Catheterization & Acute Coronary Syndrome for CCRN Review
02.14 Shock Stages for CCRN Review
02.16 Cardiogenic Shock for CCRN Review
02.17 Septic Shock for CCRN Review
03.03 Hypoglycemia for CCRN Review
06.05 Wide Complex Tachycardia for CCRN Review
ACE (angiotensin-converting enzyme) Inhibitors
Acetaminophen (Tylenol) Nursing Considerations
ACLS (Advanced cardiac life support) Drugs
Acute Coronary Syndrome (ACS)
Acute Coronary Syndrome for Certified Emergency Nursing (CEN)
Acute Inflammatory Disease (Myocarditis, Endocarditis, Pericarditis) for Progressive Care Certified Nurse (PCCN)
Acute Kidney Injury Case Study (60 min)
Acute Respiratory Distress Syndrome (ARDS) for Progressive Care Certified Nurse (PCCN)
Addicted Newborn
Adenosine (Adenocard) Nursing Considerations
Adrenal and Thyroid Disorder Emergencies for Certified Emergency Nursing (CEN)
Advanced Cardiovascular Life Support (ACLS)
Allergic Reactions and Anaphylaxis for Certified Emergency Nursing (CEN)
Amputation for Certified Emergency Nursing (CEN)
Anemia for Progressive Care Certified Nurse (PCCN)
Aneurysm and Dissection for Certified Emergency Nursing (CEN)
Angiotensin Receptor Blockers
Anti Tumor Antibiotics
Anti-Infective – Antifungals
Anti-Infective – Fluoroquinolones
Antidepressants
Antidepressants
Antineoplastics
Arterial Pressure Monitoring
Aspiration for Certified Emergency Nursing (CEN)
Atrial Dysrhythmias for Progressive Care Certified Nurse (PCCN)
Atrial Fibrillation (A Fib)
Atrial Flutter
Autonomic Nervous System (ANS)
AV Blocks Dysrhythmias for Progressive Care Certified Nurse (PCCN)
AVPU Mnemonic (The AVPU Scale)
Benzodiazepines
Blood Flow Through The Heart
Blood Pressure (BP) Control
Blood Salvage Transfusion Anticipation for Certified Perioperative Nurse (CNOR)
Blunt Thoracic Trauma
Body System Assessments
Brain Natriuretic Peptide (BNP) Lab Values
Bronchodilators
Burns for Certified Emergency Nursing (CEN)
Calcium Acetate (PhosLo) Nursing Considerations
Calcium and Magnesium Imbalance for Certified Emergency Nursing (CEN)
Calcium Carbonate (Tums) Nursing Considerations
Calcium Channel Blockers
Calcium-Ca (Hypercalcemia, Hypocalcemia)
Cardiac (Heart) Disease in Pregnancy
Cardiac (Heart) Enzymes
Cardiac (Heart) Physiology
Cardiac A&P Module Intro
Cardiac Anatomy
Cardiac Arrest Nursing Interventions for Certified Perioperative Nurse (CNOR)
Cardiac Course Introduction
Cardiac Cycle
Cardiac Glycosides
Cardiac Labs – What and When to Use Them – Live Tutoring Archive
Cardiac Labs – What and When to Use Them 2 – Live Tutoring Archive
Cardiac Stress Test
Cardiac Surgery (Post-ICU Care) for Progressive Care Certified Nurse (PCCN)
Cardiac Tamponade for Progressive Care Certified Nurse (PCCN)
Cardiac Terminology
Cardiac Valves Blood Flow Nursing Mnemonic (Toilet Paper my Ass)
Cardiac/Vascular Catheterization (Diagnostic, Interventional) for Progressive Care Certified Nurse (PCCN)
Cardiogenic Shock and Obstructive Shock for Certified Emergency Nursing (CEN)
Cardiogenic Shock For PCCN for Progressive Care Certified Nurse (PCCN)
Cardiomyopathies (Dilated, Hypertrophic, Restrictive) for Progressive Care Certified Nurse (PCCN)
Cardiopulmonary Arrest
Cardiopulmonary Arrest for Certified Emergency Nursing (CEN)
Cardiovascular Trauma for Certified Emergency Nursing (CEN)
Cerebral Metabolism
Chemotherapy Patients
Cirrhosis for Certified Emergency Nursing (CEN)
Cold Temperature-related Emergencies for Certified Emergency Nursing (CEN)
Congenital Heart Defects (CHD)
Congestive Heart Failure (CHF) Labs
Congestive Heart Failure Concept Map
Coronary Artery Disease Concept Map
Creatine Phosphokinase (CPK) Lab Values
Critical Thinking
Cushing’s Syndrome Case Study (60 min)
Day in the Life of a Med-surg Nurse
Day in the Life of a NICU Nurse
Defects of Decreased Pulmonary Blood Flow
Defects of Increased Pulmonary Blood Flow
Digoxin (Lanoxin) Nursing Considerations
Disease Specific Medications
Diuretics (Loop, Potassium Sparing, Thiazide, Furosemide/Lasix)
Dobutamine (Dobutrex) Nursing Considerations
Dopamine (Inotropin) Nursing Considerations
Dysrhythmia Emergencies
Dysrhythmias for Certified Emergency Nursing (CEN)
Dysrhythmias Labs
Echocardiogram (Cardiac Echo)
Electrical A&P of the Heart
Electrical Activity in the Heart
Electrolyte Imbalances for Progressive Care Certified Nurse (PCCN)
Electrolytes Involved in Cardiac (Heart) Conduction
Endocarditis Case Study (45 min)
Endocarditis for Certified Emergency Nursing (CEN)
Envenomation Emergencies for Certified Emergency Nursing (CEN)
Epinephrine (EpiPen) Nursing Considerations
Flight Nurse
General Anesthesia
GERD (Gastroesophageal Reflux Disease)
Goal Setting
Heart (Cardiac) and Great Vessels Assessment
Heart (Cardiac) Failure Module Intro
Heart (Cardiac) Failure Therapeutic Management
Heart (Cardiac) Sound Locations and Auscultation
Heart (Heart) Failure Exacerbation
Heart Failure (Acute Exacerbations, Chronic) for Progressive Care Certified Nurse (PCCN)
Heart Failure Case Study (45 min)
Heart Failure for Certified Emergency Nursing (CEN)
Heart Sounds Nursing Mnemonic (APE To Man – All People Enjoy Time Magazine)
Heat Temperature-related Emergencies for Certified Emergency Nursing (CEN)
Hemodynamics
Hemorrhagic Fevers for Certified Emergency Nursing (CEN)
Hiatal Hernia
Hydrocodone-Acetaminophen (Vicodin, Lortab) Nursing Considerations
Hyperemesis Gravidarum for Certified Emergency Nursing (CEN)
Hyperkalemia – Management Nursing Mnemonic (AIRED)
Hyperkalemia – Signs and Symptoms Nursing Mnemonic (Murder)
Hypertension (Uncontrolled) and Hypertensive Crisis for Progressive Care Certified Nurse (PCCN)
Hypertensive Crisis Case Study (45 min)
Hyperthyroidism Case Study (75 min)
Hypokalemia – Signs and Symptoms Nursing Mnemonic (6 L’s)
Hypoparathyroidism
Hypovolemic and Distributive Shock for Certified Emergency Nursing (CEN)
Hypovolemic Shock Case Study (OB sim) (60 min)
Influenza for Certified Emergency Nursing (CEN)
Intake and Output (I&O)
Interdisciplinary Team Participation for Certified Perioperative Nurse (CNOR)
Intraoperative Positioning
Invoicing Process
Lactate Dehydrogenase (LDH) Lab Values
Leukemia
Lorazepam (Ativan) Nursing Considerations
Lung Cancer
Magnesium-Mg (Hypomagnesemia, Hypermagnesemia)
Malignant Hyperthermia
Marfan Syndrome
Maternal Risk Factors
Meds for Postpartum Hemorrhage (PPH)
Meds for PPH (postpartum hemorrhage)
MI Surgical Intervention
Midazolam (Versed) Nursing Considerations
Minimally-Invasive Cardiac Surgery (Non-Sternal Approach) for Progressive Care Certified Nurse (PCCN)
Mixed (Cardiac) Heart Defects
Muscle Anatomy (anatomy and physiology)
Muscle Cytology
Musculoskeletal Terminology
Myocardial Infarction (MI) Case Study (45 min)
Myoglobin (MB) Lab Values
Neurogenic Shock for Certified Emergency Nursing (CEN)
Newborn Physical Exam
Noncardiac Pulmonary Edema for Certified Emergency Nursing (CEN)
Norepinephrine (Levophed) Nursing Considerations
Nursing Care and Pathophysiology for Anaphylaxis
Nursing Care and Pathophysiology for Cardiogenic Shock
Nursing Care and Pathophysiology for Cushings Syndrome
Nursing Care and Pathophysiology for Gonorrhea (STI)
Nursing Care and Pathophysiology for Heart Failure (CHF)
Nursing Care and Pathophysiology for Hyperthyroidism
Nursing Care and Pathophysiology for Hypothyroidism
Nursing Care and Pathophysiology for Hypovolemic Shock
Nursing Care and Pathophysiology for Ischemic Stroke (CVA)
Nursing Care and Pathophysiology for Myasthenia Gravis
Nursing Care and Pathophysiology for Pneumothorax & Hemothorax
Nursing Care and Pathophysiology for Pulmonary Edema
Nursing Care and Pathophysiology for SIADH (Syndrome of Inappropriate antidiuretic Hormone Secretion)
Nursing Care and Pathophysiology for Valve Disorders
Nursing Care and Pathophysiology of Acute Kidney (Renal) Injury (AKI)
Nursing Care and Pathophysiology of Angina
Nursing Care and Pathophysiology of Chronic Kidney (Renal) Disease (CKD)
Nursing Care and Pathophysiology of Coronary Artery Disease (CAD)
Nursing Care and Pathophysiology of Endocarditis and Pericarditis
Nursing Care and Pathophysiology of Hypertension (HTN)
Nursing Care and Pathophysiology of Myocardial Infarction (MI)
Nursing Care and Pathophysiology of Myocarditis