Nursing Care and Pathophysiology for Cardiogenic Shock

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Nichole Weaver
MSN/Ed,RN,CCRN
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Included In This Lesson

Study Tools For Nursing Care and Pathophysiology for Cardiogenic Shock

Shock – Signs and symptoms (Mnemonic)
Cardiogenic Shock Pathochart (Cheatsheet)
Shock (Cheatsheet)
Dopamine vs Dobutamine (Cheatsheet)
Pulmonary Artery Catheter (Image)
Cardiac Tamponade (Image)
Types of Shock (Picmonic)
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Outline

  1. Causes
    1. Myocardial infarction (MI)
    2. End-stage cardiomyopathy
    3. Papillary muscle or valve rupture
    4. Cardiac tamponade
    5. Pulmonary embolism (PE)
    6. Sudden, severe, extreme heart failure
  2.  Decreased Perfusion
    1. ↓ CO
    2. ↓ BP
    3. ↑ HR (compensation)
    4. ↑ SVR (compensation)
    5. Weak, thready pulses
    6. Cool, diaphoretic skin
    7. Pale, dusky, cyanotic, or mottled skin
    8. ↓ urine output
    9. ↓ LOC, anxiety
    10. Weakness
  3.  Volume Overload
    1. ↑ CVP
    2. JVD
    3. Pulmonary Edema
    4. Crackles
    5. Pink, frothy sputum
    6. Sudden, severe SOB
    7. Muffled Heart Sounds
    8. S3, S4 present
  4.  Therapeutic Management
    1. Treat Cause
      1. Revascularization for MI (PCI, CABG)
      2. Thrombolytics or surgical removal for PE
      3. Pericardiocentesis for cardiac tamponade
    2. Improve Contractility
      1. Dopamine – may ↑ HR
      2. Dobutamine
    3. Decrease Afterload
      1. Dobutamine
      2. Diuretics
      3. Furosemide – for Pulmonary edema
        1. Caution – may ↓ BP
    4. Surgical Intervention
      1. IABP
      2. LVAD
      3. Heart Transplant
  5.  Patient Education
    1. Health promotion for prevention of myocardial infarction (see MI lesson)
    2. Explain procedures and expectations
    3. Symptoms to report to RN or HCP
    4. Importance of positioning (HOB > 30° and legs elevated)
    5. Medication instructions, side effects

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Transcript

Okay guys, let’s talk about the basics of cardiogenic shock.

Remember the initial insult here is a broken or blocked pump. There are three main causes – myocardial infarction (remember dead heart muscle can’t pump), cardiac tamponade (which is fluid around the heart that compresses it and makes it hard to pump), and a pulmonary embolism that keeps blood from moving forward through the lungs. So the big thing we want you to see is that this is like an extreme version of heart failure. The blood can’t go forward so it backs up – so we have an increased preload. The bad pump causes our cardiac output and blood pressure to drop. The body tries to compensate by increasing the heart rate, and also by vasoconstricting and shunting blood to vital organs, so we see our afterload increase as well. What we’re left with is a pumping system that is entirely broken and vital organs that aren’t getting the blood they need. So what does this look like in our patient?

Well remember it’s like extreme, sudden, severe heart failure – so you’ve lost all perfusion forward. What happens with a lack of perfusion to the brain? Decreased LOC. Decreased perfusion to the kidneys? Decreased urine output. Decreased perfusion to the skin and extremities? Cold, pale, clammy skin, decreased pulses, slow cap refill. ALL signs of decreased perfusion. That’s your #1 clue. We may also see the problems of the blood backing up, right? Remember what happens when the blood backs up into the lungs – we get pulmonary edema and hear crackles. Then when it starts backing up into the head we see that extreme JVD – like a rope in their neck, right? So can you see how this is just sudden, extreme, severe heart failure? So keep that in mind as we move forward.

When we start thinking about treating a patient in cardiogenic shock, the most important thing is to identify and treat the cause. If they’ve had an MI, they’ll need revascularization like we talked about in the MI lesson. If it’s cardiac tamponade we need to remove the fluid from around their heart, if it’s a pulmonary embolism, we need to bust up or remove that clot. If we don’t address the cause, nothing else we do will be effective. When it comes to medication management, the big goal is to get the heart pumping more effectively and decrease the pressure it has to pump against. The top two drugs we give for patients in cardiogenic shock are dopamine and dobutamine. They will both increase contractility. Dopamine can also increase heart rate, while dobutamine can also help with vasodilation to decrease afterload. Patients who have had an MI will also still get the standard MONA treatment as well. If the patient has developed severe pulmonary edema, they may also receive a diuretic like Furosemide to offload that volume. We just have to be careful not to drop their blood pressure too much.

So, there are a lot of nursing priorities for this patient – one of which being that they need to be in an ICU. But we’re gonna focus on the top 3 concepts here. If you check out the outline and the care plan attached to this lesson, you’ll see a ton of details about specific interventions. The first concept is perfusion, we have got to monitor their hemodynamics and maintain a good cardiac output. Then, because these patients are at risk for airway and breathing issues, we need to monitor their oxygen status and intervene as needed. Then finally I added clotting because this condition might be caused by a clot in the coronary arteries or pulmonary arteries or might be because of bleeding around the heart – so we need to consider the interventions required to manage those conditions as well.

So let’s recap – cardiogenic shock is caused by a broken or blocked pump – that might be an MI, cardiac tamponade, or a PE. Most of the symptoms you see are caused by a lack of perfusion to the organs like the brain, kidneys, and skin, and by the backup of blood into the lungs and body (that’s why you see pulmonary edema and JVD). Remember it’s like a sudden, extreme version of heart failure. Treatment is focused on treating the cause, increasing contractility and getting the heart pumping more efficiently against less pressure. Our priorities are going to be perfusion and oxygenation, and then dealing with any clotting or bleeding issues depending on the cause. And finally, remember that this is an emergency, these patients need to be in an ICU and may even need to be on life support. So don’t be afraid to ask for help if you need it!

Make sure you check out the care plan and outline in this lesson to see lots of details about nursing care and interventions. We love you guys! Happy nursing!

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Concepts Covered:

  • Basics of Human Biology
  • Renal Disorders
  • Intraoperative Nursing
  • Preoperative Nursing
  • Perioperative Nursing Roles
  • Basics of NCLEX
  • Test Taking Strategies
  • Concepts of Population Health
  • Respiratory System
  • Endocrine System
  • Urinary System
  • Communication
  • Oncologic Disorders
  • Fundamentals of Emergency Nursing
  • Prioritization
  • Delegation
  • Emotions and Motivation
  • Documentation and Communication
  • Eating Disorders
  • Respiratory Disorders
  • Noninfectious Respiratory Disorder
  • Shock
  • Disorders of Pancreas
  • Neurological Emergencies
  • Central Nervous System Disorders – Brain
  • Emergency Care of the Neurological Patient
  • Circulatory System
  • Cardiac Disorders
  • Emergency Care of the Cardiac Patient
  • Hematologic Disorders
  • Hematologic Disorders
  • Medication Administration

Study Plan Lessons

Homeostasis
Nursing Care and Pathophysiology for Rhabdomyolysis
Malignant Hyperthermia
Intubation in the OR
Preoperative (Preop)Assessment
Perioperative Nursing Roles
Purpose of Nursing Care Plans
Continuity of Care
Disasters & Bioterrorism
Practice Settings
Breathing Movements
Breathing Control
Respiratory Functions of Blood
Thyroid Gland
Pituitary Gland
Pancreas
Adrenal Gland
Renal (Kidney) Acid-Base Balance
Formation & Excretion of Urine
Renal (Kidney) Structure & Function
Renal (Kidney) Fluid & Electrolyte Balance
Respiratory Structure & Function
Communicating with Other Departments
Confidence in Communication
Communicating with Patients
Communicating with Family Members
Communicating with UAPs
Communicating with Other Nurses
Communicating with Providers
Giving Handoff Report
Leukemia
Pediatric Oncology Basics
Anion Gap
Triage
Prioritization
Delegation
Maslow’s Hierarchy of Needs in Nursing
Handoff Report
SBAR Communication
Admissions, Discharges, and Transfers
Potassium-K (Hyperkalemia, Hypokalemia)
ABGs Nursing Normal Lab Values
ABG (Arterial Blood Gas) Interpretation-The Basics
ROME – ABG (Arterial Blood Gas) Interpretation
ABGs Tic-Tac-Toe interpretation Method
Respiratory Acidosis (interpretation and nursing interventions)
Respiratory Alkalosis
Metabolic Acidosis (interpretation and nursing diagnosis)
Metabolic Alkalosis
Lactic Acid
Base Excess & Deficit
Nursing Care and Pathophysiology of Diabetic Ketoacidosis (DKA)
Oxygen Delivery Module Intro
Hierarchy of O2 Delivery
Artificial Airways
Vent Alarms
Stroke Assessment (CVA)
Seizure Therapeutic Management
Nursing Care and Pathophysiology for Seizure
Nursing Care and Pathophysiology for Cardiogenic Shock
Normal Sinus Rhythm
Sinus Bradycardia
Sinus Tachycardia
Atrial Flutter
Atrial Fibrillation (A Fib)
Premature Atrial Contraction (PAC)
Supraventricular Tachycardia (SVT)
Premature Ventricular Contraction (PVC)
Ventricular Tachycardia (V-tach)
Ventricular Fibrillation (V Fib)
1st Degree AV Heart Block
2nd Degree AV Heart Block Type 1 (Mobitz I, Wenckebach)
2nd Degree AV Heart Block Type 2 (Mobitz II)
3rd Degree AV Heart Block (Complete Heart Block)
Sickle Cell Anemia
Hemophilia
Epoetin Alfa
6 Rights of Medication Administration